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Blood|August 31, 2006
Inhibition of overactivated p38 MAPK can restore hematopoiesis in myelodysplastic syndrome progenitorsTony A Navas, Mani Mohindru, Myka Estes, et al.
JCI Insight|February 8, 2023
Mitochondrial and NAD+ metabolism predict recovery from acute kidney injury in a diverse mouse populationJean-David Morel, Maroun Bou Sleiman, Terytty Yang Li, et al.
British Journal of Pharmacology|May 20, 2015
Prolyl hydroxylase inhibition corrects functional iron deficiency and inflammation-induced anaemia in ratsTerrance D Barrett, Heather L Palomino, Theresa I Brondstetter, et al.
The Journal of Experimental Medicine|February 14, 2023
The genetic background shapes the susceptibility to mitochondrial dysfunction and NASH progressionGiorgia Benegiamo, Giacomo V G von Alvensleben, Sandra Rodríguez-López, et al.
Molecular Pharmacology|May 19, 2006
Transforming growth factor-beta receptor type 1 (TGFbetaRI) kinase activity but not p38 activation is required for TGFbetaRI-induced myofibroblast differentiation and profibrotic gene expressionAnn M Kapoun, Nicholas J Gaspar, Ying Wang, et al.
Neuro-Oncology|May 25, 2007
Inhibiting TGF-beta signaling restores immune surveillance in the SMA-560 glioma modelThomas-Toan Tran, Martin Uhl, Jing Ying Ma, et al.
Blood|May 14, 2008
Inhibition of the TGF-beta receptor I kinase promotes hematopoiesis in MDSLi Zhou, Aaron N Nguyen, Davendra Sohal, et al.
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