Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Proceedings of the National Academy of Sciences of the United States of America|February 9, 2005
Artemis-independent functions of DNA-dependent protein kinase in Ig heavy chain class switch recombination and developmentSean Rooney, Frederick W Alt, JoAnn Sekiguchi, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 5, 2008
Complementary functions of ATM and H2AX in development and suppression of genomic instabilityShan Zha, JoAnn Sekiguchi, James W Brush, et al.
The Journal of Experimental Medicine|April 8, 2009
Impact of a hypomorphic Artemis disease allele on lymphocyte development, DNA end processing, and genome stabilityYing Huang, William Giblin, Martina Kubec, et al.
Nature|March 9, 2007
Endonuclease-independent LINE-1 retrotransposition at mammalian telomeresTammy A Morrish, José Luis Garcia-Perez, Thomas D Stamato, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 26, 2004
Artemis and p53 cooperate to suppress oncogenic N-myc amplification in progenitor B cellsSean Rooney, JoAnn Sekiguchi, Scott Whitlow, et al.
The Journal of Experimental Medicine|March 5, 2003
Defective DNA repair and increased genomic instability in Artemis-deficient murine cellsSean Rooney, Frederick W Alt, David Lombard, et al.
The Journal of Experimental Medicine|October 29, 2003
Impaired V(D)J recombination and lymphocyte development in core RAG1-expressing miceDarryll D Dudley, JoAnn Sekiguchi, Chengming Zhu, et al.
Molecular Cell|December 31, 2002
Leaky Scid phenotype associated with defective V(D)J coding end processing in Artemis-deficient miceSean Rooney, JoAnn Sekiguchi, Chengming Zhu, et al.
Molecular and Cellular Biology|April 1, 2009
Structural and functional interaction between the human DNA repair proteins DNA ligase IV and XRCC4Peï-Yu Wu, Philippe Frit, SriLakshmi Meesala, et al.
Blood|January 8, 2009
Leaky severe combined immunodeficiency and aberrant DNA rearrangements due to a hypomorphic RAG1 mutationWilliam Giblin, Monalisa Chatterji, Gerwin Westfield, et al.
Pageof 2