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American Journal of Physiology. Heart and Circulatory Physiology|August 9, 2020
Enhanced dimethylarginine degradation improves coronary flow reserve and exercise tolerance in Duchenne muscular dystrophy carrier miceJoanne F Garbincius, Lauren E Merz, Ashley J Cuttitta, et al.
JACC. Basic to Translational Science|September 1, 2021
Molecular Signature of HFpEF: Systems Biology in a Cardiac-Centric Large Animal ModelAndrew A Gibb, Emma K Murray, Deborah M Eaton, et al.
The EMBO Journal|May 22, 2026
Genetic ablation of neuronal mitochondrial calcium uptake impedes Alzheimer's disease progressionPooja Jadiya, Elena Berezhnaya, Devin W Kolmetzky, et al.
Research Square|July 18, 2025
MICU proteins facilitate Ca2+-dependent mitochondrial metabolon formation to regulate cellular energetics - independent of MCUHenry M Cohen, Benjamin Gottschalk, Carmen Choya-Foces, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
YAP1 and QSER1 are Key Modulators of Embryonic Signaling Pathways in the Mammalian EpiblastElizabeth Abraham, Thomas Roule, Aidan Douglas, et al.
EMBO Reports|March 27, 2026
YAP1 and QSER1 are key modulators of embryonic signaling pathways in the mammalian epiblastElizabeth Abraham, Thomas Roule, Aidan Douglas, et al.
Biorxiv : the Preprint Server for Biology|October 24, 2023
TMEM65 regulates NCLX-dependent mitochondrial calcium effluxJoanne F Garbincius, Oniel Salik, Henry M Cohen, et al.
Nature Metabolism|April 8, 2025
TMEM65 regulates and is required for NCLX-dependent mitochondrial calcium effluxJoanne F Garbincius, Oniel Salik, Henry M Cohen, et al.
Nature Metabolism|May 13, 2026
MICU proteins facilitate calcium-dependent mitochondrial metabolon formation to regulate cellular energetics independently of MCUHenry M Cohen, Benjamin Gottschalk, Carmen Choya-Foces, et al.
Circulation Research|October 23, 2020
Interaction of the Joining Region in Junctophilin-2 With the L-Type Ca2+ Channel Is Pivotal for Cardiac Dyad Assembly and Intracellular Ca2+ DynamicsPolina Gross, Jaslyn Johnson, Carlos M Romero, et al.
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