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Molecular Genetics and Metabolism|March 7, 2018
Chronic diazoxide treatment decreases fat mass and improves endurance capacity in an obese mouse model of Prader-Willi syndromeJocelyn M Bischof, Rachel WevrickPhysiological Genomics|May 5, 2005
Genome-wide analysis of gene transcription in the hypothalamusJocelyn M Bischof, Rachel WevrickDevelopmental Dynamics : an Official Publication of the American Association of Anatomists|October 28, 2003
A MAGE/NDN-like gene in zebrafishJocelyn M Bischof, Marc Ekker, Rachel WevrickHuman Molecular Genetics|August 31, 2007
Inactivation of the mouse Magel2 gene results in growth abnormalities similar to Prader-Willi syndromeJocelyn M Bischof, Colin L Stewart, Rachel WevrickBritish Journal of Pharmacology|June 25, 2016
Magel2-null mice are hyper-responsive to setmelanotide, a melanocortin 4 receptor agonistJocelyn M Bischof, Lex H T Van Der Ploeg, William F Colmers, et al.Plos One|April 22, 2020
A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1K Vanessa Carias, Mercedes Zoeteman, Abigail Seewald, et al.American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|February 10, 2009
Regionally reduced brain volume, altered serotonin neurochemistry, and abnormal behavior in mice null for the circadian rhythm output gene Magel2Rebecca E Mercer, Erin M Kwolek, Jocelyn M Bischof, et al.Human Molecular Genetics|July 21, 2016
Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromesAin A Kamaludin, Christa Smolarchuk, Jocelyn M Bischof, et al.Pageof 1