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Cell
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May 12, 2015
Disruptions of topological chromatin domains cause pathogenic rewiring of gene-enhancer interactions
Darío G Lupiáñez, Katerina Kraft, Verena Heinrich, et al.
Nature Communications
|
December 24, 2014
European sea bass genome and its variation provide insights into adaptation to euryhalinity and speciation
Mbaye Tine, Heiner Kuhl, Pierre-Alexandre Gagnaire, et al.
Brain : a Journal of Neurology
|
January 25, 2014
Sensory neuropathy with bone destruction due to a mutation in the membrane-shaping atlastin GTPase 3
Uwe Kornak, Inès Mademan, Marte Schinke, et al.
American Journal of Human Genetics
|
January 21, 2014
Mutations in PGAP3 impair GPI-anchor maturation, causing a subtype of hyperphosphatasia with mental retardation
Malcolm F Howard, Yoshiko Murakami, Alistair T Pagnamenta, et al.
Science Translational Medicine
|
September 5, 2014
Effective diagnosis of genetic disease by computational phenotype analysis of the disease-associated genome
Tomasz Zemojtel, Sebastian Köhler, Luisa Mackenroth, et al.
American Journal of Human Genetics
|
December 7, 2014
Homozygous and compound-heterozygous mutations in TGDS cause Catel-Manzke syndrome
Nadja Ehmke, Almuth Caliebe, Rainer Koenig, et al.
Leukemia
|
March 8, 2019
PAX5 biallelic genomic alterations define a novel subgroup of B-cell precursor acute lymphoblastic leukemia
Lorenz Bastian, Michael P Schroeder, Cornelia Eckert, et al.
The Journal of Experimental Medicine
|
February 27, 2013
Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome
Daniel Kotlarz, Natalia Ziętara, Gulbu Uzel, et al.
Nature Genetics
|
August 31, 2010
Identity-by-descent filtering of exome sequence data identifies PIGV mutations in hyperphosphatasia mental retardation syndrome
Peter M Krawitz, Michal R Schweiger, Christian Rödelsperger, et al.
American Journal of Human Genetics
|
November 4, 2017
De Novo Mutations in SLC25A24 Cause a Craniosynostosis Syndrome with Hypertrichosis, Progeroid Appearance, and Mitochondrial Dysfunction
Nadja Ehmke, Luitgard Graul-Neumann, Lukasz Smorag, et al.
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of 9
Search research articles
Search
Showing results (71-80 of 87) with videos related to
Sort By:
Page
of 9
Cell
|
May 12, 2015
Disruptions of topological chromatin domains cause pathogenic rewiring of gene-enhancer interactions
Darío G Lupiáñez, Katerina Kraft, Verena Heinrich, et al.
Nature Communications
|
December 24, 2014
European sea bass genome and its variation provide insights into adaptation to euryhalinity and speciation
Mbaye Tine, Heiner Kuhl, Pierre-Alexandre Gagnaire, et al.
Brain : a Journal of Neurology
|
January 25, 2014
Sensory neuropathy with bone destruction due to a mutation in the membrane-shaping atlastin GTPase 3
Uwe Kornak, Inès Mademan, Marte Schinke, et al.
American Journal of Human Genetics
|
January 21, 2014
Mutations in PGAP3 impair GPI-anchor maturation, causing a subtype of hyperphosphatasia with mental retardation
Malcolm F Howard, Yoshiko Murakami, Alistair T Pagnamenta, et al.
Science Translational Medicine
|
September 5, 2014
Effective diagnosis of genetic disease by computational phenotype analysis of the disease-associated genome
Tomasz Zemojtel, Sebastian Köhler, Luisa Mackenroth, et al.
American Journal of Human Genetics
|
December 7, 2014
Homozygous and compound-heterozygous mutations in TGDS cause Catel-Manzke syndrome
Nadja Ehmke, Almuth Caliebe, Rainer Koenig, et al.
Leukemia
|
March 8, 2019
PAX5 biallelic genomic alterations define a novel subgroup of B-cell precursor acute lymphoblastic leukemia
Lorenz Bastian, Michael P Schroeder, Cornelia Eckert, et al.
The Journal of Experimental Medicine
|
February 27, 2013
Loss-of-function mutations in the IL-21 receptor gene cause a primary immunodeficiency syndrome
Daniel Kotlarz, Natalia Ziętara, Gulbu Uzel, et al.
Nature Genetics
|
August 31, 2010
Identity-by-descent filtering of exome sequence data identifies PIGV mutations in hyperphosphatasia mental retardation syndrome
Peter M Krawitz, Michal R Schweiger, Christian Rödelsperger, et al.
American Journal of Human Genetics
|
November 4, 2017
De Novo Mutations in SLC25A24 Cause a Craniosynostosis Syndrome with Hypertrichosis, Progeroid Appearance, and Mitochondrial Dysfunction
Nadja Ehmke, Luitgard Graul-Neumann, Lukasz Smorag, et al.
Page
of 9