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The Journal of Biological Chemistry|October 23, 2012
Human mismatch repair protein hMutLα is required to repair short slipped-DNAs of trinucleotide repeatsGagan B Panigrahi, Meghan M Slean, Jodie P Simard, et al.Proceedings of the National Academy of Sciences of the United States of America|June 24, 2010
Isolated short CTG/CAG DNA slip-outs are repaired efficiently by hMutSbeta, but clustered slip-outs are poorly repairedGagan B Panigrahi, Meghan M Slean, Jodie P Simard, et al.Journal of Cell Science|June 23, 2011
Overexpression of HSP70 inhibits cofilin phosphorylation and promotes lymphocyte migration in heat-stressed cellsJodie P Simard, Danielle N Reynolds, Alan P Kraguljac, et al.Human Molecular Genetics|October 12, 2010
Huntington's and myotonic dystrophy hESCs: down-regulated trinucleotide repeat instability and mismatch repair machinery expression upon differentiationAnna Seriola, Claudia Spits, Jodie P Simard, et al.DNA Repair|December 12, 2012
Tissue-specific mismatch repair protein expression: MSH3 is higher than MSH6 in multiple mouse tissuesStéphanie Tomé, Jodie P Simard, Meghan M Slean, et al.Human Molecular Genetics|November 6, 2013
Expression levels of DNA replication and repair genes predict regional somatic repeat instability in the brain but are not altered by polyglutamine disease protein expression or ageAmanda G Mason, Stephanie Tomé, Jodie P Simard, et al.Plos Genetics|March 8, 2013
MSH3 polymorphisms and protein levels affect CAG repeat instability in Huntington's disease miceStéphanie Tomé, Kevin Manley, Jodie P Simard, et al.Disease Models & Mechanisms|November 9, 2012
Neurons and cardiomyocytes derived from induced pluripotent stem cells as a model for mitochondrial defects in Friedreich's ataxiaAurore Hick, Marie Wattenhofer-Donzé, Satyan Chintawar, et al.Pageof 1