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The Journal of Biological Chemistry|January 17, 2015
RNA toxicity and missplicing in the common eye disease fuchs endothelial corneal dystrophyJintang Du, Ross A Aleff, Elisabetta Soragni, et al.Proceedings of the National Academy of Sciences of the United States of America|October 3, 2008
The HDAC inhibitor 4b ameliorates the disease phenotype and transcriptional abnormalities in Huntington's disease transgenic miceElizabeth A Thomas, Giovanni Coppola, Paula A Desplats, et al.Annals of Neurology|December 14, 2011
A gene expression phenotype in lymphocytes from Friedreich ataxia patientsGiovanni Coppola, Ryan Burnett, Susan Perlman, et al.Neurobiology of Disease|May 17, 2012
Histone deacetylase (HDAC) inhibitors targeting HDAC3 and HDAC1 ameliorate polyglutamine-elicited phenotypes in model systems of Huntington's diseaseHaiqun Jia, Judit Pallos, Vincent Jacques, et al.Annals of Neurology|August 28, 2014
Epigenetic therapy for Friedreich ataxiaElisabetta Soragni, Wenyan Miao, Marco Iudicello, et al.Biorxiv : the Preprint Server for Biology|March 27, 2026
Friedreich ataxia transcriptomic dysregulation and identification of cell type-specific biomarkers: A systematic review and meta-analysisMarnie L Maddock, Sara Miellet, Anjila Dongol, et al.Frontiers in Molecular Biosciences|September 22, 2022
A non-synonymous single nucleotide polymorphism in SIRT6 predicts neurological severity in Friedreich ataxiaLayne N Rodden, Christian Rummey, Yi Na Dong, et al.Nature Chemical Biology|December 8, 2009
Reduced histone deacetylase 7 activity restores function to misfolded CFTR in cystic fibrosisDarren M Hutt, David Herman, Ana P C Rodrigues, et al.Pageof 6