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Biochemistry and Biophysics Reports|October 9, 2024
Depletion of the Rho GTPases Cdc42, Rac1 or RhoA reduces PDGF-induced STAT1 and STAT3 signalingErik Wåhlén, Johan Lennartsson, Johan HeldinCellular Signalling|May 23, 2022
Differential impact of lipid raft depletion on platelet-derived growth factor (PDGF)-induced ERK1/2 MAP-kinase, SRC and AKT signalingErik Wåhlén, Frida Olsson, Ola Söderberg, et al.Experimental Cell Research|February 16, 2017
The atypical Rho GTPase RhoD is a regulator of actin cytoskeleton dynamics and directed cell migrationMagdalena Blom, Katarina Reis, Johan Heldin, et al.Frontiers in Neuroscience|March 20, 2013
Interactions of zebrafish peptide YYb with the neuropeptide Y-family receptors Y4, Y7, Y8a, and Y8bGörel Sundström, Tomas A Larsson, Bo Xu, et al.Nucleic Acids Research|January 23, 2024
Visualizing DNA single- and double-strand breaks in the Flash comet assay by DNA polymerase-assisted end-labellingErik Bivehed, Björn Hellman, Leonie Wenson, et al.Biochemical and Biophysical Research Communications|October 24, 2024
Crosstalk between 1,25(OH)<sub>2</sub>-Vitamin D<sub>3</sub> and the growth factors EGF and PDGF-BB: Impact on CYP24A1 expression and cell proliferationFrida Olsson, Erik Wåhlén, Johan Heldin, et al.Biochemical and Biophysical Research Communications|May 10, 2023
Activated EGFR and PDGFR internalize in separate vesicles and downstream AKT and ERK1/2 signaling are differentially impacted by cholesterol depletionErik Wåhlén, Frida Olsson, Doroteya Raykova, et al.Expert Review of Proteomics|February 16, 2024
The method developer's guide to oligonucleotide designLeonie Wenson, Mattias Leino, Malin Jarvius, et al.Experimental Cell Research|April 11, 2019
Dynamin inhibitors impair platelet-derived growth factor β-receptor dimerization and signalingJohan Heldin, Marie Rubin Sander, Mattias Leino, et al.The Journal of Biological Chemistry|May 14, 2011
Exocyst complex component 3-like 2 (EXOC3L2) associates with the exocyst complex and mediates directional migration of endothelial cellsIrmeli Barkefors, Peder Fredlund Fuchs, Johan Heldin, et al.Pageof 3