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Physiological Genomics|October 4, 2002
Angiotensin II is associated with activation of NF-kappaB-mediated genes and downregulation of PPARsDoris M Tham, Baby Martin-McNulty, Yi-xin Wang, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|November 19, 2020
The effects of diet composition and chronic obesity on muscle growth and functionLuís G O de Sousa, Andrea G Marshall, Jennifer E Norman, et al.
Circulation Research|January 20, 2007
Triglyceride-rich lipoproteins prime aortic endothelium for an enhanced inflammatory response to tumor necrosis factor-alphaHarold J Ting, James P Stice, Ulrich Y Schaff, et al.
Journal of Lipid Research|December 24, 2005
Interaction between mild hypercholesterolemia, HDL-cholesterol levels, and angiotensin II in intimal hyperplasia in miceValdeci da Cunha, Baby Martin-McNulty, Jon Vincelette, et al.
Journal of Cellular and Molecular Medicine|September 17, 2009
Imatinib inhibits vascular smooth muscle proteoglycan synthesis and reduces LDL binding in vitro and aortic lipid deposition in vivoMandy L Ballinger, Narin Osman, Kazuhiko Hashimura, et al.
Plos One|December 5, 2012
Postprandial apoE isoform and conformational changes associated with VLDL lipolysis products modulate monocyte inflammationLaura J den Hartigh, Robin Altman, Romobia Hutchinson, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|October 22, 2002
Angiotensin II injures the arterial wall causing increased aortic stiffening in apolipoprotein E-deficient miceDoris M Tham, Baby Martin-McNulty, Yi-Xin Wang, et al.
Metabolic Syndrome and Related Disorders|February 17, 2016
Effects of Nonpurified and Choline Supplemented or Nonsupplemented Purified Diets on Hepatic Steatosis and Methionine Metabolism in C3H MiceRaisa Syed, Noreene M Shibata, Kusum K Kharbanda, et al.
Journal of Lipid Research|December 8, 2009
VLDL lipolysis products increase VLDL fluidity and convert apolipoprotein E4 into a more expanded conformationSarada D Tetali, Madhu S Budamagunta, Catalina Simion, et al.
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