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Arthritis Research & Therapy|May 19, 2007
Pro-apoptotic Bid is required for the resolution of the effector phase of inflammatory arthritisJohn C Scatizzi, Jack Hutcheson, Emily Bickel, et al.Arthritis and Rheumatism|September 30, 2006
Bim deficiency leads to exacerbation and prolongation of joint inflammation in experimental arthritisJohn C Scatizzi, Emily Bickel, Jack Hutcheson, et al.Inflammation|November 6, 2004
IL-4 and IL-10 inhibition of spontaneous monocyte apoptosis is associated with Flip upregulationJoy Eslick, John C Scatizzi, Lee Albee, et al.Journal of Immunology (Baltimore, Md. : 1950)|February 29, 2012
The Lbw2 locus promotes autoimmune hemolytic anemiaJohn C Scatizzi, Maria K Haraldsson, K Michael Pollard, et al.The Journal of Experimental Medicine|June 22, 2005
Combined loss of proapoptotic genes Bak or Bax with Bim synergizes to cause defects in hematopoiesis and in thymocyte apoptosisJack Hutcheson, John C Scatizzi, Emily Bickel, et al.Clinical Immunology (Orlando, Fla.)|December 3, 2014
The effect of the autoimmunity-associated gene, PTPN22, on a BXSB-derived model of lupusChristian J Maine, Kristi Marquardt, John C Scatizzi, et al.Plos One|November 22, 2011
Systemic autoimmunity and lymphoproliferation are associated with excess IL-7 and inhibited by IL-7Rα blockadeRosana Gonzalez-Quintial, Brian R Lawson, John C Scatizzi, et al.The American Journal of Pathology|May 3, 2006
p21Cip1 is required for the development of monocytes and their response to serum transfer-induced arthritisJohn C Scatizzi, Jack Hutcheson, Emily Bickel, et al.Immunity|February 16, 2008
Combined deficiency of proapoptotic regulators Bim and Fas results in the early onset of systemic autoimmunityJack Hutcheson, John C Scatizzi, Akbar M Siddiqui, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 9, 2004
Fas death receptor signaling represses monocyte numbers and macrophage activation in vivoNathaniel J Brown, Jack Hutcheson, Emily Bickel, et al.Pageof 2