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John C Shryock

Showing results (1-10 of 52) with videos related to

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American Journal of Physiology. Heart and Circulatory Physiology|August 25, 2009
A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytesYejia Song, John C Shryock, Luiz Belardinelli
American Journal of Physiology. Heart and Circulatory Physiology|March 4, 2008
An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytesYejia Song, John C Shryock, Luiz Belardinelli
Pharmacology & Therapeutics|July 30, 2008
Pathophysiology and pharmacology of the cardiac "late sodium current."Antonio Zaza, Luiz Belardinelli, John C Shryock
Channels (Austin, Tex.)|December 17, 2008
Block of tetrodotoxin-sensitive, Na(V)1.7 and tetrodotoxin-resistant, Na(V)1.8, Na+ channels by ranolazineSridharan Rajamani, John C Shryock, Luiz Belardinelli
Journal of Cardiovascular Pharmacology|June 4, 2008
Rapid kinetic interactions of ranolazine with HERG K+ currentSridharan Rajamani, John C Shryock, Luiz Belardinelli
Circulation|January 5, 2002
Selective attenuation of isoproterenol-stimulated arrhythmic activity by a partial agonist of adenosine A1 receptorYejia Song, Lin Wu, John C Shryock, et al.
Journal of Cardiovascular Pharmacology|July 10, 2004
Antagonism by ranolazine of the pro-arrhythmic effects of increasing late INa in guinea pig ventricular myocytesYejia Song, John C Shryock, Lin Wu, et al.
Heart Rhythm|October 29, 2005
Use of preclinical assays to predict risk of drug-induced torsades de pointesLuiz Belardinelli, John C Shryock, Lin Wu, et al.
The Journal of Pharmacology and Experimental Therapeutics|October 20, 2005
An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit heartsLin Wu, John C Shryock, Yejia Song, et al.
The Journal of Pharmacology and Experimental Therapeutics|May 1, 2009
The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunctionYuzhi Wu, Yejia Song, Luiz Belardinelli, et al.
Pageof 6

Showing results (1-10 of 52) with videos related to

Sort By:
Pageof 6
American Journal of Physiology. Heart and Circulatory Physiology|August 25, 2009
A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytesYejia Song, John C Shryock, Luiz Belardinelli
American Journal of Physiology. Heart and Circulatory Physiology|March 4, 2008
An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytesYejia Song, John C Shryock, Luiz Belardinelli
Pharmacology & Therapeutics|July 30, 2008
Pathophysiology and pharmacology of the cardiac "late sodium current."Antonio Zaza, Luiz Belardinelli, John C Shryock
Channels (Austin, Tex.)|December 17, 2008
Block of tetrodotoxin-sensitive, Na(V)1.7 and tetrodotoxin-resistant, Na(V)1.8, Na+ channels by ranolazineSridharan Rajamani, John C Shryock, Luiz Belardinelli
Journal of Cardiovascular Pharmacology|June 4, 2008
Rapid kinetic interactions of ranolazine with HERG K+ currentSridharan Rajamani, John C Shryock, Luiz Belardinelli
Circulation|January 5, 2002
Selective attenuation of isoproterenol-stimulated arrhythmic activity by a partial agonist of adenosine A1 receptorYejia Song, Lin Wu, John C Shryock, et al.
Journal of Cardiovascular Pharmacology|July 10, 2004
Antagonism by ranolazine of the pro-arrhythmic effects of increasing late INa in guinea pig ventricular myocytesYejia Song, John C Shryock, Lin Wu, et al.
Heart Rhythm|October 29, 2005
Use of preclinical assays to predict risk of drug-induced torsades de pointesLuiz Belardinelli, John C Shryock, Lin Wu, et al.
The Journal of Pharmacology and Experimental Therapeutics|October 20, 2005
An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit heartsLin Wu, John C Shryock, Yejia Song, et al.
The Journal of Pharmacology and Experimental Therapeutics|May 1, 2009
The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunctionYuzhi Wu, Yejia Song, Luiz Belardinelli, et al.
Pageof 6