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The Journal of Clinical Investigation|March 26, 2010
The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channelsBoyi Liu, John E Linley, Xiaona Du, et al.
The Journal of Pathology|October 20, 2011
Potential role of reduced basolateral potassium (IKCa3.1) channel expression in the pathogenesis of diarrhoea in ulcerative colitisAdel Al-Hazza, John E Linley, Qadeer Aziz, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 16, 2012
Reactive oxygen species are second messengers of neurokinin signaling in peripheral sensory neuronsJohn E Linley, Lezanne Ooi, Louisa Pettinger, et al.
The Journal of Steroid Biochemistry and Molecular Biology|December 23, 2006
Role of protein kinase C in aldosterone-induced non-genomic inhibition of basolateral potassium channels in human colonic cryptsKate A Bowley, John E Linley, Gerrard G Robins, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 8, 2010
Transcriptional control of KCNQ channel genes and the regulation of neuronal excitabilityMariusz Mucha, Lezanne Ooi, John E Linley, et al.
Pain Reports|April 23, 2026
Monoclonal antibody inhibition of PAR2 reduces phenotype severity and pain in murine inflammatory bowel diseaseAnne Ritoux, Laura J Grundy, Pravallika Manjappa, et al.
Brain : a Journal of Neurology|September 11, 2012
Neurological perspectives on voltage-gated sodium channelsNiels Eijkelkamp, John E Linley, Mark D Baker, et al.
Peptides|March 26, 2013
Potency optimization of Huwentoxin-IV on hNav1.7: a neurotoxin TTX-S sodium-channel antagonist from the venom of the Chinese bird-eating spider Selenocosmia huwenaJefferson D Revell, Per-Eric Lund, John E Linley, et al.
Plos One|June 3, 2015
Regulation of Nav1.7: A Conserved SCN9A Natural Antisense Transcript Expressed in Dorsal Root GangliaJennifer Koenig, Robert Werdehausen, John E Linley, et al.
Neuromolecular Medicine|January 8, 2013
Novel mutations mapping to the fourth sodium channel domain of Nav1.7 result in variable clinical manifestations of primary erythromelalgiaRoman Cregg, Bisola Laguda, Robert Werdehausen, et al.
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