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John H B Bridge

Showing results (1-10 of 25) with videos related to

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Circulation Research|March 1, 2003
Ca2+ sparks in rabbit ventricular myocytes evoked by action potentials: involvement of clusters of L-type Ca2+ channelsMasashi Inoue, John H B Bridge
Biophysical Journal|August 23, 2005
Variability in couplon size in rabbit ventricular myocytesMasashi Inoue, John H B Bridge
Biophysical Journal|September 11, 2007
Revealing the cellular basis of heart failureJohn H B Bridge, Eleonora Savio
Circulation Research|May 10, 2008
What are the consequences of phosphorylation and hyperphosphorylation of ryanodine receptors in normal and failing heart?John H B Bridge, Eleonora Savio-Galimberti
Cardiovascular Research|April 11, 2017
Mechanisms regulating Ca2+ release in cardiomyocytesJohn H B Bridge, Natalia S Torres
Circulation. Heart Failure|October 8, 2009
Loss of intracellular and intercellular synchrony of calcium release in systolic heart failureJoshua I Goldhaber, John H B Bridge
The Journal of General Physiology|October 27, 2010
Challenging quantal calcium signaling in cardiac myocytesAlexandra Zahradníková, Marta Gaburjáková, John H B Bridge, et al.
Biophysical Journal|May 18, 2011
Strain transfer in ventricular cardiomyocytes to their transverse tubular system revealed by scanning confocal microscopyThomas G McNary, John H B Bridge, Frank B Sachse
The Journal of Physiology|August 19, 2008
New insights into the structure and function of couplonsJohn H B Bridge, Natalia S Torres, Eric A Sobie
Circulation Research|March 4, 2006
A novel mechanism of pacemaker control that depends on high levels of cAMP and PKA-dependent phosphorylation: a precisely controlled biological clockJohn H B Bridge, Christopher J Davidson, Eleonora Savio-Galimberti
Pageof 3

Showing results (1-10 of 25) with videos related to

Sort By:
Pageof 3
Circulation Research|March 1, 2003
Ca2+ sparks in rabbit ventricular myocytes evoked by action potentials: involvement of clusters of L-type Ca2+ channelsMasashi Inoue, John H B Bridge
Biophysical Journal|August 23, 2005
Variability in couplon size in rabbit ventricular myocytesMasashi Inoue, John H B Bridge
Biophysical Journal|September 11, 2007
Revealing the cellular basis of heart failureJohn H B Bridge, Eleonora Savio
Circulation Research|May 10, 2008
What are the consequences of phosphorylation and hyperphosphorylation of ryanodine receptors in normal and failing heart?John H B Bridge, Eleonora Savio-Galimberti
Cardiovascular Research|April 11, 2017
Mechanisms regulating Ca2+ release in cardiomyocytesJohn H B Bridge, Natalia S Torres
Circulation. Heart Failure|October 8, 2009
Loss of intracellular and intercellular synchrony of calcium release in systolic heart failureJoshua I Goldhaber, John H B Bridge
The Journal of General Physiology|October 27, 2010
Challenging quantal calcium signaling in cardiac myocytesAlexandra Zahradníková, Marta Gaburjáková, John H B Bridge, et al.
Biophysical Journal|May 18, 2011
Strain transfer in ventricular cardiomyocytes to their transverse tubular system revealed by scanning confocal microscopyThomas G McNary, John H B Bridge, Frank B Sachse
The Journal of Physiology|August 19, 2008
New insights into the structure and function of couplonsJohn H B Bridge, Natalia S Torres, Eric A Sobie
Circulation Research|March 4, 2006
A novel mechanism of pacemaker control that depends on high levels of cAMP and PKA-dependent phosphorylation: a precisely controlled biological clockJohn H B Bridge, Christopher J Davidson, Eleonora Savio-Galimberti
Pageof 3