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John H B Bridge

Showing results (11-20 of 25) with videos related to

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Biophysical Journal|January 29, 2008
Allosteric activation of Na+-Ca2+ exchange by L-type Ca2+ current augments the trigger flux for SR Ca2+ release in ventricular myocytesEric A Sobie, Mark B Cannell, John H B Bridge
Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing|February 13, 2009
Towards computational modeling of excitation-contraction coupling in cardiac myocytes: reconstruction of structures and proteins from confocal imagingFrank B Sachse, Eleonora Savio-Galimberti, Joshua I Goldhaber, et al.
Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing|January 31, 2008
Sub-micrometer anatomical models of the sarcolemma of cardiac myocytes based on confocal imagingFrank B Sachse, Eleonora Savio-Galimberti, Joshua I Goldhaber, et al.
The Journal of Physiology|September 15, 2010
Na+ currents are required for efficient excitation-contraction coupling in rabbit ventricular myocytes: a possible contribution of neuronal Na+ channelsNatalia S Torres, Robert Larbig, Alex Rock, et al.
Journal of Molecular and Cellular Cardiology|June 18, 2013
Na/Ca exchange and contraction of the heartMichela Ottolia, Natalia Torres, John H B Bridge, et al.
The Journal of Physiology|July 21, 2010
Activation of reverse Na+-Ca2+ exchange by the Na+ current augments the cardiac Ca2+ transient: evidence from NCX knockout miceRobert Larbig, Natalia Torres, John H B Bridge, et al.
Circulation. Heart Failure|August 22, 2015
Cardiac Resynchronization Therapy Reduces Subcellular Heterogeneity of Ryanodine Receptors, T-Tubules, and Ca2+ Sparks Produced by Dyssynchronous Heart FailureHui Li, Justin G Lichter, Thomas Seidel, et al.
Circulation Research|February 19, 2005
Metabolic inhibition alters subcellular calcium release patterns in rat ventricular myocytes: implications for defective excitation-contraction coupling during cardiac ischemia and failureGary H Fukumoto, Scott T Lamp, Christi Motter, et al.
Progress in Biophysics and Molecular Biology|August 14, 2012
Mechanical modulation of the transverse tubular system of ventricular cardiomyocytesThomas G McNary, Kenneth W Spitzer, Hilary Holloway, et al.
Biophysical Journal|August 5, 2010
Sodium-calcium exchange is essential for effective triggering of calcium release in mouse heartPatricia Neco, Beth Rose, Nhi Huynh, et al.
Pageof 3

Showing results (11-20 of 25) with videos related to

Sort By:
Pageof 3
Biophysical Journal|January 29, 2008
Allosteric activation of Na+-Ca2+ exchange by L-type Ca2+ current augments the trigger flux for SR Ca2+ release in ventricular myocytesEric A Sobie, Mark B Cannell, John H B Bridge
Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing|February 13, 2009
Towards computational modeling of excitation-contraction coupling in cardiac myocytes: reconstruction of structures and proteins from confocal imagingFrank B Sachse, Eleonora Savio-Galimberti, Joshua I Goldhaber, et al.
Pacific Symposium on Biocomputing. Pacific Symposium on Biocomputing|January 31, 2008
Sub-micrometer anatomical models of the sarcolemma of cardiac myocytes based on confocal imagingFrank B Sachse, Eleonora Savio-Galimberti, Joshua I Goldhaber, et al.
The Journal of Physiology|September 15, 2010
Na+ currents are required for efficient excitation-contraction coupling in rabbit ventricular myocytes: a possible contribution of neuronal Na+ channelsNatalia S Torres, Robert Larbig, Alex Rock, et al.
Journal of Molecular and Cellular Cardiology|June 18, 2013
Na/Ca exchange and contraction of the heartMichela Ottolia, Natalia Torres, John H B Bridge, et al.
The Journal of Physiology|July 21, 2010
Activation of reverse Na+-Ca2+ exchange by the Na+ current augments the cardiac Ca2+ transient: evidence from NCX knockout miceRobert Larbig, Natalia Torres, John H B Bridge, et al.
Circulation. Heart Failure|August 22, 2015
Cardiac Resynchronization Therapy Reduces Subcellular Heterogeneity of Ryanodine Receptors, T-Tubules, and Ca2+ Sparks Produced by Dyssynchronous Heart FailureHui Li, Justin G Lichter, Thomas Seidel, et al.
Circulation Research|February 19, 2005
Metabolic inhibition alters subcellular calcium release patterns in rat ventricular myocytes: implications for defective excitation-contraction coupling during cardiac ischemia and failureGary H Fukumoto, Scott T Lamp, Christi Motter, et al.
Progress in Biophysics and Molecular Biology|August 14, 2012
Mechanical modulation of the transverse tubular system of ventricular cardiomyocytesThomas G McNary, Kenneth W Spitzer, Hilary Holloway, et al.
Biophysical Journal|August 5, 2010
Sodium-calcium exchange is essential for effective triggering of calcium release in mouse heartPatricia Neco, Beth Rose, Nhi Huynh, et al.
Pageof 3