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Cancer Research|September 10, 2009
The receptor tyrosine kinase EPHB4 has tumor suppressor activities in intestinal tumorigenesisHiginio Dopeso, Silvia Mateo-Lozano, Rocco Mazzolini, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|September 19, 2013
Molecular imaging of death receptor 5 occupancy and saturation kinetics in vivo by humanized monoclonal antibody CS-1008Ingrid J G Burvenich, Fook T Lee, Glenn A Cartwright, et al.
Cancer Research|November 17, 2005
Mechanisms of inactivation of the receptor tyrosine kinase EPHB2 in colorectal tumorsHafid Alazzouzi, Veronica Davalos, Antti Kokko, et al.
Molecular Cancer Therapeutics|March 18, 2016
Dual Targeting of Bromodomain and Extraterminal Domain Proteins, and WNT or MAPK Signaling, Inhibits c-MYC Expression and Proliferation of Colorectal Cancer CellsLars Tögel, Rebecca Nightingale, Anderly C Chueh, et al.
Molecular Cancer Therapeutics|February 10, 2021
Rapid Resistance of FGFR-driven Gastric Cancers to Regorafenib and Targeted FGFR Inhibitors can be Overcome by Parallel Inhibition of MEKDavid K Lau, Ian Y Luk, Laura J Jenkins, et al.
British Journal of Cancer|December 6, 2017
Mechanisms of inactivation of the tumour suppressor gene RHOA in colorectal cancerHiginio Dopeso, Paulo Rodrigues, Josipa Bilic, et al.
Oral Oncology|July 4, 2012
Phase II trial of the histone deacetylase inhibitor romidepsin in patients with recurrent/metastatic head and neck cancerMissak Haigentz, Mimi Kim, Catherine Sarta, et al.
Scientific Reports|January 31, 2018
DUSP5 is methylated in CIMP-high colorectal cancer but is not a major regulator of intestinal cell proliferation and tumorigenesisLars Tögel, Rebecca Nightingale, Rui Wu, et al.
Development (Cambridge, England)|June 28, 2021
EHF is essential for epidermal and colonic epithelial homeostasis, and suppresses Apc-initiated colonic tumorigenesisCamilla M Reehorst, Rebecca Nightingale, Ian Y Luk, et al.
Cancer Research|January 14, 2010
Apoptotic sensitivity of colon cancer cells to histone deacetylase inhibitors is mediated by an Sp1/Sp3-activated transcriptional program involving immediate-early gene inductionAndrew J Wilson, Anderly C Chueh, Lars Tögel, et al.
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