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Cell Cycle (Georgetown, Tex.)|December 4, 2002
Induction of transcription through the p300 CRD1 motif by p21WAF1/CIP1 is core promoter specific and cyclin dependent kinase independentDavid J Gregory, Elisa Garcia-Wilson, Jason C Poole, et al.
The Biochemical Journal|May 25, 2002
A novel form of the RelA nuclear factor kappaB subunit is induced by and forms a complex with the proto-oncogene c-MycNeil R Chapman, Gill A Webster, Peter J Gillespie, et al.
BMC Microbiology|March 30, 2019
Induction of the immunoprotective coat of Yersinia pestis at body temperature is mediated by the Caf1R transcription factorAbdulmajeed D Al-Jawdah, Iglika G Ivanova, Helen Waller, et al.
Molecular Biology of the Cell|July 9, 2011
The role of RelA (p65) threonine 505 phosphorylation in the regulation of cell growth, survival, and migrationAichi Msaki, Ana M Sánchez, Li Fang Koh, et al.
BMC Molecular Biology|August 10, 2004
The p68 and p72 DEAD box RNA helicases interact with HDAC1 and repress transcription in a promoter-specific mannerBrian J Wilson, Gaynor J Bates, Samantha M Nicol, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 30, 2014
A 9,000-year-old caribou hunting structure beneath Lake HuronJohn M O'Shea, Ashley K Lemke, Elizabeth P Sonnenburg, et al.
Biochimica Et Biophysica Acta|January 15, 2008
IEX-1 directly interferes with RelA/p65 dependent transactivation and regulation of apoptosisAlexander Arlt, Philip Rosenstiel, Marie-Luise Kruse, et al.
The Journal of Biological Chemistry|January 6, 2005
SIRT1 deacetylation and repression of p300 involves lysine residues 1020/1024 within the cell cycle regulatory domain 1Toula Bouras, Maofu Fu, Anthony A Sauve, et al.
Communications Biology|December 9, 2021
Centrosome dysfunction associated with somatic expression of the synaptonemal complex protein TEX12Sumit Sandhu, Ieng F Sou, Jill E Hunter, et al.
Molecular Cell|April 30, 2003
P300 transcriptional repression is mediated by SUMO modificationDavid Girdwood, Donna Bumpass, Owen A Vaughan, et al.
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