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John P Pribis

Showing results (11-20 of 18) with videos related to

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Science Advances|June 19, 2021
Two mechanisms of chromosome fragility at replication-termination sites in bacteriaQian Mei, Devon M Fitzgerald, Jingjing Liu, et al.
Science Advances|June 23, 2023
Drugging evolution of antibiotic resistance at a regulatory network hubYin Zhai, John P Pribis, Sean W Dooling, et al.
Molecular Medicine (Cambridge, Mass.)|September 9, 2015
The HIV Protease Inhibitor Saquinavir Inhibits HMGB1-Driven Inflammation by Targeting the Interaction of Cathepsin V with TLR4/MyD88John P Pribis, Yousef Al-Abed, Huan Yang, et al.
Molecular Cell|April 6, 2019
Gamblers: An Antibiotic-Induced Evolvable Cell Subpopulation Differentiated by Reactive-Oxygen-Induced General Stress ResponseJohn P Pribis, Libertad García-Villada, Yin Zhai, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|October 31, 2015
HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4Jingjing Cai, Hong Yuan, Qingde Wang, et al.
Science Advances|January 17, 2017
Holliday junction trap shows how cells use recombination and a junction-guardian role of RecQ helicaseJun Xia, Li-Tzu Chen, Qian Mei, et al.
The Journal of Experimental Medicine|January 7, 2015
MD-2 is required for disulfide HMGB1-dependent TLR4 signalingHuan Yang, Haichao Wang, Zhongliang Ju, et al.
Cell|January 12, 2019
Bacteria-to-Human Protein Networks Reveal Origins of Endogenous DNA DamageJun Xia, Li-Ya Chiu, Ralf B Nehring, et al.
Pageof 2

Showing results (11-20 of 18) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 18 results.
Science Advances|June 19, 2021
Two mechanisms of chromosome fragility at replication-termination sites in bacteriaQian Mei, Devon M Fitzgerald, Jingjing Liu, et al.
Science Advances|June 23, 2023
Drugging evolution of antibiotic resistance at a regulatory network hubYin Zhai, John P Pribis, Sean W Dooling, et al.
Molecular Medicine (Cambridge, Mass.)|September 9, 2015
The HIV Protease Inhibitor Saquinavir Inhibits HMGB1-Driven Inflammation by Targeting the Interaction of Cathepsin V with TLR4/MyD88John P Pribis, Yousef Al-Abed, Huan Yang, et al.
Molecular Cell|April 6, 2019
Gamblers: An Antibiotic-Induced Evolvable Cell Subpopulation Differentiated by Reactive-Oxygen-Induced General Stress ResponseJohn P Pribis, Libertad García-Villada, Yin Zhai, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|October 31, 2015
HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4Jingjing Cai, Hong Yuan, Qingde Wang, et al.
Science Advances|January 17, 2017
Holliday junction trap shows how cells use recombination and a junction-guardian role of RecQ helicaseJun Xia, Li-Tzu Chen, Qian Mei, et al.
The Journal of Experimental Medicine|January 7, 2015
MD-2 is required for disulfide HMGB1-dependent TLR4 signalingHuan Yang, Haichao Wang, Zhongliang Ju, et al.
Cell|January 12, 2019
Bacteria-to-Human Protein Networks Reveal Origins of Endogenous DNA DamageJun Xia, Li-Ya Chiu, Ralf B Nehring, et al.
Pageof 2