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John Ravits

Showing results (71-80 of 102) with videos related to

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Acta Neuropathologica|June 9, 2018
Transcriptome-pathology correlation identifies interplay between TDP-43 and the expression of its kinase CK1E in sporadic ALSFlorian Krach, Ranjan Batra, Emily C Wheeler, et al.
Nature Neuroscience|January 16, 2019
Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegenerationZe'ev Melamed, Jone López-Erauskin, Michael W Baughn, et al.
Science Translational Medicine|October 25, 2013
Targeting RNA foci in iPSC-derived motor neurons from ALS patients with a C9ORF72 repeat expansionDhruv Sareen, Jacqueline G O'Rourke, Pratap Meera, et al.
Biorxiv : the Preprint Server for Biology|December 11, 2023
Somatic Mosaicism in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia Reveals Widespread Degeneration from Focal MutationsZinan Zhou, Junho Kim, August Yue Huang, et al.
The Lancet. Neurology|February 19, 2013
Controversies and priorities in amyotrophic lateral sclerosisMartin R Turner, Orla Hardiman, Michael Benatar, et al.
Human Molecular Genetics|February 8, 2018
Elongator subunit 3 (ELP3) modifies ALS through tRNA modificationAndre Bento-Abreu, Gunilla Jager, Bart Swinnen, et al.
Iscience|November 3, 2023
FUS-dependent microRNA deregulations identify TRIB2 as a druggable target for ALS motor neuronsWan Yun Ho, Li-Ling Chak, Jin-Hui Hor, et al.
Molecular Neurodegeneration|February 14, 2026
Impaired nucleocytoplasmic transport in SOD1-mediated ALSShirel Argueti-Ostrovsky, Su Min Lim, Olubankole A Arogundade, et al.
Nature Neuroscience|December 19, 2018
TDP-43 extracted from frontotemporal lobar degeneration subject brains displays distinct aggregate assemblies and neurotoxic effects reflecting disease progression ratesFlorent Laferrière, Zuzanna Maniecka, Manuela Pérez-Berlanga, et al.
Annals of Neurology|May 1, 2007
Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutationsIan R A Mackenzie, Eileen H Bigio, Paul G Ince, et al.
Pageof 11

Showing results (71-80 of 102) with videos related to

Sort By:
Pageof 11
Acta Neuropathologica|June 9, 2018
Transcriptome-pathology correlation identifies interplay between TDP-43 and the expression of its kinase CK1E in sporadic ALSFlorian Krach, Ranjan Batra, Emily C Wheeler, et al.
Nature Neuroscience|January 16, 2019
Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegenerationZe'ev Melamed, Jone López-Erauskin, Michael W Baughn, et al.
Science Translational Medicine|October 25, 2013
Targeting RNA foci in iPSC-derived motor neurons from ALS patients with a C9ORF72 repeat expansionDhruv Sareen, Jacqueline G O'Rourke, Pratap Meera, et al.
Biorxiv : the Preprint Server for Biology|December 11, 2023
Somatic Mosaicism in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia Reveals Widespread Degeneration from Focal MutationsZinan Zhou, Junho Kim, August Yue Huang, et al.
The Lancet. Neurology|February 19, 2013
Controversies and priorities in amyotrophic lateral sclerosisMartin R Turner, Orla Hardiman, Michael Benatar, et al.
Human Molecular Genetics|February 8, 2018
Elongator subunit 3 (ELP3) modifies ALS through tRNA modificationAndre Bento-Abreu, Gunilla Jager, Bart Swinnen, et al.
Iscience|November 3, 2023
FUS-dependent microRNA deregulations identify TRIB2 as a druggable target for ALS motor neuronsWan Yun Ho, Li-Ling Chak, Jin-Hui Hor, et al.
Molecular Neurodegeneration|February 14, 2026
Impaired nucleocytoplasmic transport in SOD1-mediated ALSShirel Argueti-Ostrovsky, Su Min Lim, Olubankole A Arogundade, et al.
Nature Neuroscience|December 19, 2018
TDP-43 extracted from frontotemporal lobar degeneration subject brains displays distinct aggregate assemblies and neurotoxic effects reflecting disease progression ratesFlorent Laferrière, Zuzanna Maniecka, Manuela Pérez-Berlanga, et al.
Annals of Neurology|May 1, 2007
Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutationsIan R A Mackenzie, Eileen H Bigio, Paul G Ince, et al.
Pageof 11