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Cell Reports|July 21, 2021
Homeostatic apoptosis prevents competition-induced atrophy in follicular B cellsStéphane Chappaz, Kate McArthur, Liam Kealy, et al.Cell Death and Differentiation|December 19, 2023
Combined absence of TRP53 target genes ZMAT3, PUMA and p21 cause a high incidence of cancer in miceMargs S Brennan, Kerstin Brinkmann, Gerard Romero Sola, et al.Virus Research|September 30, 2009
Dysregulation of hepatocyte cell cycle and cell viability by hepatitis B virusRuth Chin, Ulrich Nachbur, Linda Earnest-Silveira, et al.Gastroenterology|June 23, 2020
Loss of NFKB1 Results in Expression of Tumor Necrosis Factor and Activation of Signal Transducer and Activator of Transcription 1 to Promote Gastric Tumorigenesis in MiceJun T Low, Michael Christie, Matthias Ernst, et al.Diabetes|December 5, 2009
Glucose induces pancreatic islet cell apoptosis that requires the BH3-only proteins Bim and Puma and multi-BH domain protein BaxMark D McKenzie, Emma Jamieson, Elisa S Jansen, et al.Cell Death and Differentiation|September 30, 2017
Coordinated repression of BIM and PUMA by Epstein-Barr virus latent genes maintains the survival of Burkitt lymphoma cellsLeah Fitzsimmons, Andrew J Boyce, Wenbin Wei, et al.Blood|April 23, 2009
The histone deacetylase inhibitors LAQ824 and LBH589 do not require death receptor signaling or a functional apoptosome to mediate tumor cell death or therapeutic efficacyLeigh Ellis, Michael Bots, Ralph K Lindemann, et al.Cell Reports|February 3, 2016
The Transcription Factor ASCIZ and Its Target DYNLL1 Are Essential for the Development and Expansion of MYC-Driven B Cell LymphomaDavid M Wong, Lingli Li, Sabine Jurado, et al.Proceedings of the National Academy of Sciences of the United States of America|May 8, 2007
Two molecular pathways initiate mitochondria-dependent dopaminergic neurodegeneration in experimental Parkinson's diseaseCeline Perier, Jordi Bové, Du-Chu Wu, et al.The Journal of Cell Biology|April 3, 2002
The anti-apoptotic activity of XIAP is retained upon mutation of both the caspase 3- and caspase 9-interacting sitesJohn Silke, Christine J Hawkins, Paul G Ekert, et al.Pageof 46