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John V Walsh

Showing results (1-10 of 15) with videos related to

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Science (New York, N.Y.)|January 10, 2009
The time to demand fundingCraig C Mello, John V Walsh
The Journal of General Physiology|February 28, 2002
Natural bile acids and synthetic analogues modulate large conductance Ca2+-activated K+ (BKCa) channel activity in smooth muscle cellsAlejandro M Dopico, John V Walsh, Joshua J Singer
The Journal of General Physiology|June 27, 2002
Spontaneous transient outward currents arise from microdomains where BK channels are exposed to a mean Ca(2+) concentration on the order of 10 microM during a Ca(2+) sparkRonghua Zhuge, Kevin E Fogarty, Richard A Tuft, et al.
American Journal of Physiology. Cell Physiology|November 1, 2002
Site of action of fatty acids and other charged lipids on BKCa channels from arterial smooth muscle cellsAlison L Clarke, Steven Petrou, John V Walsh, et al.
American Journal of Physiology. Cell Physiology|October 10, 2002
Modulation of BK(Ca) channel activity by fatty acids: structural requirements and mechanism of actionAlison L Clarke, Steven Petrou, John V Walsh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 13, 2009
Individual calcium syntillas do not trigger spontaneous exocytosis from nerve terminals of the neurohypophysisJames M McNally, Valérie De Crescenzo, Kevin E Fogarty, et al.
Biophysical Journal|October 29, 2003
Quantitative analysis of spontaneous mitochondrial depolarizationsCatherine M O'Reilly, Kevin E Fogarty, Robert M Drummond, et al.
American Journal of Physiology. Cell Physiology|April 13, 2004
Spontaneous mitochondrial depolarizations are independent of SR Ca2+ releaseCatherine M O'Reilly, Kevin E Fogarty, Robert M Drummond, et al.
The Journal of Physiology|August 17, 2014
Catecholamine exocytosis during low frequency stimulation in mouse adrenal chromaffin cells is primarily asynchronous and controlled by the novel mechanism of Ca2+ syntilla suppressionJason J Lefkowitz, Valerie DeCrescenzo, Kailai Duan, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 29, 2011
Type 1 ryanodine receptor knock-in mutation causing central core disease of skeletal muscle also displays a neuronal phenotypeValerie De Crescenzo, Kevin E Fogarty, Jason J Lefkowitz, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Science (New York, N.Y.)|January 10, 2009
The time to demand fundingCraig C Mello, John V Walsh
The Journal of General Physiology|February 28, 2002
Natural bile acids and synthetic analogues modulate large conductance Ca2+-activated K+ (BKCa) channel activity in smooth muscle cellsAlejandro M Dopico, John V Walsh, Joshua J Singer
The Journal of General Physiology|June 27, 2002
Spontaneous transient outward currents arise from microdomains where BK channels are exposed to a mean Ca(2+) concentration on the order of 10 microM during a Ca(2+) sparkRonghua Zhuge, Kevin E Fogarty, Richard A Tuft, et al.
American Journal of Physiology. Cell Physiology|November 1, 2002
Site of action of fatty acids and other charged lipids on BKCa channels from arterial smooth muscle cellsAlison L Clarke, Steven Petrou, John V Walsh, et al.
American Journal of Physiology. Cell Physiology|October 10, 2002
Modulation of BK(Ca) channel activity by fatty acids: structural requirements and mechanism of actionAlison L Clarke, Steven Petrou, John V Walsh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 13, 2009
Individual calcium syntillas do not trigger spontaneous exocytosis from nerve terminals of the neurohypophysisJames M McNally, Valérie De Crescenzo, Kevin E Fogarty, et al.
Biophysical Journal|October 29, 2003
Quantitative analysis of spontaneous mitochondrial depolarizationsCatherine M O'Reilly, Kevin E Fogarty, Robert M Drummond, et al.
American Journal of Physiology. Cell Physiology|April 13, 2004
Spontaneous mitochondrial depolarizations are independent of SR Ca2+ releaseCatherine M O'Reilly, Kevin E Fogarty, Robert M Drummond, et al.
The Journal of Physiology|August 17, 2014
Catecholamine exocytosis during low frequency stimulation in mouse adrenal chromaffin cells is primarily asynchronous and controlled by the novel mechanism of Ca2+ syntilla suppressionJason J Lefkowitz, Valerie DeCrescenzo, Kailai Duan, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 29, 2011
Type 1 ryanodine receptor knock-in mutation causing central core disease of skeletal muscle also displays a neuronal phenotypeValerie De Crescenzo, Kevin E Fogarty, Jason J Lefkowitz, et al.
Pageof 2