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The Journal of Biological Chemistry|December 19, 2009
GSK-3 represses growth factor-inducible genes by inhibiting NF-kappaB in quiescent cellsJulie R Graham, John W Tullai, Geoffrey M CooperCell Cycle (Georgetown, Tex.)|September 9, 2011
A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cellsJohn W Tullai, Julie R Graham, Geoffrey M CooperThe Journal of Biological Chemistry|February 11, 2004
Identification of transcription factor binding sites upstream of human genes regulated by the phosphatidylinositol 3-kinase and MEK/ERK signaling pathwaysJohn W Tullai, Michael E Schaffer, Steven Mullenbrock, et al.Plos One|June 8, 2011
AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cellsJohn W Tullai, Silvia Tacheva, Laura J Owens, et al.The Journal of Biological Chemistry|February 6, 2007
Glycogen synthase kinase-3 represses cyclic AMP response element-binding protein (CREB)-targeted immediate early genes in quiescent cellsJohn W Tullai, Jie Chen, Michael E Schaffer, et al.The Journal of Biological Chemistry|June 19, 2007
Immediate-early and delayed primary response genes are distinct in function and genomic architectureJohn W Tullai, Michael E Schaffer, Steven Mullenbrock, et al.BMC Cell Biology|January 30, 2008
Phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of FOXO and non-canonical activation of NFkappaB transcription factorsJolyon Terragni, Julie R Graham, Kenneth W Adams, et al.Pageof 1