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Jonathan E Tullis

Showing results (1-10 of 12) with videos related to

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Iscience|May 22, 2023
Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulationJonathan E Tullis, K Ulrich Bayer
Scientific Reports|July 9, 2024
Induction of LTP mechanisms in dually innervated dendritic spinesJonathan E Tullis, K Ulrich Bayer
Journal of Neurophysiology|April 21, 2021
Young DAPK1 knockout mice have altered presynaptic functionDayton J Goodell, Jonathan E Tullis, K Ulrich Bayer
Plos One|July 28, 2020
The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targetingJonathan E Tullis, Nicole L Rumian, Carolyn Nicole Brown, et al.
Iscience|May 27, 2022
Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling eventsCarolyn Nicole Brown, Nicole L Rumian, Jonathan E Tullis, et al.
Brain Research|October 23, 2021
CaMKIIα knockout protects from ischemic neuronal cell death after resuscitation from cardiac arrestNicole L Rumian, Nicholas E Chalmers, Jonathan E Tullis, et al.
Iscience|October 27, 2021
GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTPJonathan E Tullis, Olivia R Buonarati, Steven J Coultrap, et al.
Cell Reports|October 12, 2024
LTP expression mediated by autonomous activity of GluN2B-bound CaMKIINicole L Rumian, C Madison Barker, Matthew E Larsen, et al.
Cell Reports|January 9, 2020
CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac ArrestOlivia R Buonarati, Sarah G Cook, Dayton J Goodell, et al.
Nature|August 30, 2023
LTP induction by structural rather than enzymatic functions of CaMKIIJonathan E Tullis, Matthew E Larsen, Nicole L Rumian, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Iscience|May 22, 2023
Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulationJonathan E Tullis, K Ulrich Bayer
Scientific Reports|July 9, 2024
Induction of LTP mechanisms in dually innervated dendritic spinesJonathan E Tullis, K Ulrich Bayer
Journal of Neurophysiology|April 21, 2021
Young DAPK1 knockout mice have altered presynaptic functionDayton J Goodell, Jonathan E Tullis, K Ulrich Bayer
Plos One|July 28, 2020
The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targetingJonathan E Tullis, Nicole L Rumian, Carolyn Nicole Brown, et al.
Iscience|May 27, 2022
Aβ-induced synaptic impairments require CaMKII activity that is stimulated by indirect signaling eventsCarolyn Nicole Brown, Nicole L Rumian, Jonathan E Tullis, et al.
Brain Research|October 23, 2021
CaMKIIα knockout protects from ischemic neuronal cell death after resuscitation from cardiac arrestNicole L Rumian, Nicholas E Chalmers, Jonathan E Tullis, et al.
Iscience|October 27, 2021
GluN2B S1303 phosphorylation by CaMKII or DAPK1: no indication for involvement in ischemia or LTPJonathan E Tullis, Olivia R Buonarati, Steven J Coultrap, et al.
Cell Reports|October 12, 2024
LTP expression mediated by autonomous activity of GluN2B-bound CaMKIINicole L Rumian, C Madison Barker, Matthew E Larsen, et al.
Cell Reports|January 9, 2020
CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac ArrestOlivia R Buonarati, Sarah G Cook, Dayton J Goodell, et al.
Nature|August 30, 2023
LTP induction by structural rather than enzymatic functions of CaMKIIJonathan E Tullis, Matthew E Larsen, Nicole L Rumian, et al.
Pageof 2