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Jonathan H Jaggar

Showing results (11-20 of 96) with videos related to

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The Journal of Biological Chemistry|November 25, 2010
Caveolin-1 assembles type 1 inositol 1,4,5-trisphosphate receptors and canonical transient receptor potential 3 channels into a functional signaling complex in arterial smooth muscle cellsAdebowale Adebiyi, Damodaran Narayanan, Jonathan H Jaggar
Elife|March 24, 2022
STIMulating blood pressureTessa A C Garrud, Jonathan H Jaggar
American Journal of Physiology. Heart and Circulatory Physiology|June 12, 2004
The permissive role of endothelial NO in CO-induced cerebrovascular dilationEbrahim Barkoudah, Jonathan H Jaggar, Charles W Leffler
American Journal of Physiology. Heart and Circulatory Physiology|April 19, 2011
Carbon monoxide as an endogenous vascular modulatorCharles W Leffler, Helena Parfenova, Jonathan H Jaggar
Molecular Pharmacology|May 31, 2008
Sulfonylurea receptor-dependent and -independent pathways mediate vasodilation induced by ATP-sensitive K+ channel openersAdebowale Adebiyi, Elizabeth M McNally, Jonathan H Jaggar
Circulation Research|July 16, 2005
Mitochondria-derived reactive oxygen species dilate cerebral arteries by activating Ca2+ sparksQi Xi, Sergei Y Cheranov, Jonathan H Jaggar
American Journal of Physiology. Heart and Circulatory Physiology|July 26, 2011
Vasodilation induced by oxygen/glucose deprivation is attenuated in cerebral arteries of SUR2 null miceAdebowale Adebiyi, Elizabeth M McNally, Jonathan H Jaggar
Circulation Research|July 12, 2017
Endothelin-1 Stimulates Vasoconstriction Through Rab11A Serine 177 PhosphorylationXue Zhai, M Dennis Leo, Jonathan H Jaggar
The Journal of Physiology|December 14, 2016
Angiotensin II reduces the surface abundance of K<sub>V</sub> 1.5 channels in arterial myocytes to stimulate vasoconstrictionMichael W Kidd, Simon Bulley, Jonathan H Jaggar
The Journal of Biological Chemistry|July 9, 2011
CaV1.3 channels and intracellular calcium mediate osmotic stress-induced N-terminal c-Jun kinase activation and disruption of tight junctions in Caco-2 CELL MONOLAYERSGeetha Samak, Damodaran Narayanan, Jonathan H Jaggar, et al.
Pageof 10

Showing results (11-20 of 96) with videos related to

Sort By:
Pageof 10
The Journal of Biological Chemistry|November 25, 2010
Caveolin-1 assembles type 1 inositol 1,4,5-trisphosphate receptors and canonical transient receptor potential 3 channels into a functional signaling complex in arterial smooth muscle cellsAdebowale Adebiyi, Damodaran Narayanan, Jonathan H Jaggar
Elife|March 24, 2022
STIMulating blood pressureTessa A C Garrud, Jonathan H Jaggar
American Journal of Physiology. Heart and Circulatory Physiology|June 12, 2004
The permissive role of endothelial NO in CO-induced cerebrovascular dilationEbrahim Barkoudah, Jonathan H Jaggar, Charles W Leffler
American Journal of Physiology. Heart and Circulatory Physiology|April 19, 2011
Carbon monoxide as an endogenous vascular modulatorCharles W Leffler, Helena Parfenova, Jonathan H Jaggar
Molecular Pharmacology|May 31, 2008
Sulfonylurea receptor-dependent and -independent pathways mediate vasodilation induced by ATP-sensitive K+ channel openersAdebowale Adebiyi, Elizabeth M McNally, Jonathan H Jaggar
Circulation Research|July 16, 2005
Mitochondria-derived reactive oxygen species dilate cerebral arteries by activating Ca2+ sparksQi Xi, Sergei Y Cheranov, Jonathan H Jaggar
American Journal of Physiology. Heart and Circulatory Physiology|July 26, 2011
Vasodilation induced by oxygen/glucose deprivation is attenuated in cerebral arteries of SUR2 null miceAdebowale Adebiyi, Elizabeth M McNally, Jonathan H Jaggar
Circulation Research|July 12, 2017
Endothelin-1 Stimulates Vasoconstriction Through Rab11A Serine 177 PhosphorylationXue Zhai, M Dennis Leo, Jonathan H Jaggar
The Journal of Physiology|December 14, 2016
Angiotensin II reduces the surface abundance of K<sub>V</sub> 1.5 channels in arterial myocytes to stimulate vasoconstrictionMichael W Kidd, Simon Bulley, Jonathan H Jaggar
The Journal of Biological Chemistry|July 9, 2011
CaV1.3 channels and intracellular calcium mediate osmotic stress-induced N-terminal c-Jun kinase activation and disruption of tight junctions in Caco-2 CELL MONOLAYERSGeetha Samak, Damodaran Narayanan, Jonathan H Jaggar, et al.
Pageof 10