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The British Journal of Dermatology|February 17, 2024
Acceptability of 'as needed' biologic therapy in psoriasis: insights from a multistakeholder mixed-methods studyDavid Gleeson, Maneeha Naveed, Lucy Moorhead, et al.
The Journal of Investigative Dermatology|April 9, 2013
The IL23R A/Gln381 allele promotes IL-23 unresponsiveness in human memory T-helper 17 cells and impairs Th17 responses in psoriasis patientsPaola Di Meglio, Federica Villanova, Luca Napolitano, et al.
JAMA Dermatology|March 29, 2018
Comparison of Drug Discontinuation, Effectiveness, and Safety Between Clinical Trial Eligible and Ineligible Patients in BADBIRKayleigh J Mason, Jonathan N W N Barker, Catherine H Smith, et al.
The Journal of Investigative Dermatology|September 23, 2006
Null mutations in the filaggrin gene (FLG) determine major susceptibility to early-onset atopic dermatitis that persists into adulthoodJonathan N W N Barker, Colin N A Palmer, Yiwei Zhao, et al.
The Journal of Investigative Dermatology|December 31, 2005
Interaction between genetic control of vascular endothelial growth factor production and retinoid responsiveness in psoriasisHelen S Young, Angela M Summers, Ian R Read, et al.
The Journal of Investigative Dermatology|June 9, 2015
Differential Drug Survival of Biologic Therapies for the Treatment of Psoriasis: A Prospective Observational Cohort Study from the British Association of Dermatologists Biologic Interventions Register (BADBIR)Richard B Warren, Catherine H Smith, Zenas Z N Yiu, et al.
NPJ Digital Medicine|September 27, 2023
Systematic review of deep learning image analyses for the diagnosis and monitoring of skin diseaseShern Ping Choy, Byung Jin Kim, Alexandra Paolino, et al.
The Journal of Investigative Dermatology|March 22, 2003
Genetic analysis of PSORS1 distinguishes guttate psoriasis and palmoplantar pustulosisKati Asumalahti, Mahreen Ameen, Sari Suomela, et al.
The Journal of Investigative Dermatology|April 6, 2007
Filaggrin null alleles are not associated with psoriasisYiwei Zhao, Ana Terron-Kwiatkowski, Haihui Liao, et al.
The Journal of Allergy and Clinical Immunology|September 9, 2008
Analysis of the individual and aggregate genetic contributions of previously identified serine peptidase inhibitor Kazal type 5 (SPINK5), kallikrein-related peptidase 7 (KLK7), and filaggrin (FLG) polymorphisms to eczema riskStephan Weidinger, Hansjörg Baurecht, Stefan Wagenpfeil, et al.
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