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Proceedings of the National Academy of Sciences of the United States of America|November 7, 2022
Cryo-EM structures of cancer-specific helical and kinase domain mutations of PI3KαXiao Liu, Qingtong Zhou, Jonathan R Hart, et al.Current Topics in Microbiology and Immunology|June 29, 2010
Phosphatidylinositol 3-kinase: the oncoproteinPeter K Vogt, Jonathan R Hart, Marco Gymnopoulos, et al.Oncotarget|October 20, 2014
In vivo quantification and perturbation of Myc-Max interactions and the impact on oncogenic potentialPhilipp Raffeiner, Ruth Röck, Andrea Schraffl, et al.Viruses|January 24, 2019
The Importance of Being Non-Defective: A Mini Review Dedicated to the Memory of Jan SvobodaPeter K VogtCancer Cell|August 12, 2008
Drug-resistant phosphatidylinositol 3-kinase: guidance for the preemptive strikePeter K VogtCell Cycle (Georgetown, Tex.)|December 17, 2009
Hot-spot mutations in p110alpha of phosphatidylinositol 3-kinase (pI3K): differential interactions with the regulatory subunit p85 and with RASLi Zhao, Peter K VogtProceedings of the National Academy of Sciences of the United States of America|February 13, 2008
Helical domain and kinase domain mutations in p110alpha of phosphatidylinositol 3-kinase induce gain of function by different mechanismsLi Zhao, Peter K VogtCell Cycle (Georgetown, Tex.)|February 12, 2011
Understanding PLZF: two transcriptional targets, REDD1 and smooth muscle α-actin, define new questions in growth control, senescence, self-renewal and tumor suppressionMarina Kolesnichenko, Peter K VogtPageof 11