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The Journal of Biological Chemistry
|
August 10, 2007
Plasma membrane targeting is essential for Rem-mediated Ca2+ channel inhibition
Robert N Correll, Chunyan Pang, Brian S Finlin, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
September 16, 2015
Loss of Rad-GTPase produces a novel adaptive cardiac phenotype resistant to systolic decline with aging
Janet R Manning, Catherine N Withers, Bryana Levitan, et al.
The Journal of Biological Chemistry
|
June 23, 2006
Analysis of the complex between Ca2+ channel beta-subunit and the Rem GTPase
Brian S Finlin, Robert N Correll, Chunyan Pang, et al.
Stem Cells and Development
|
May 31, 2008
In vitro electrophysiological drug testing using human embryonic stem cell derived cardiomyocytes
Oren Caspi, Ilanit Itzhaki, Izhak Kehat, et al.
Cell Calcium
|
February 21, 2009
Steady-state coupling of plasma membrane calcium entry to extrusion revealed by novel L-type calcium channel block
William C Lester, Elizabeth A Schroder, Don E Burgess, et al.
Journal of Molecular and Cellular Cardiology
|
February 8, 2021
Rad-GTPase contributes to heart rate via L-type calcium channel regulation
Bryana M Levitan, Brooke M Ahern, Ajoy Aloysius, et al.
Channels (Austin, Tex.)
|
May 12, 2010
Rem GTPase interacts with the proximal CaV1.2 C-terminus and modulates calcium-dependent channel inactivation
Chunyan Pang, Shawn M Crump, Ling Jin, et al.
Bone
|
July 23, 2017
Rad GTPase is essential for the regulation of bone density and bone marrow adipose tissue in mice
Catherine N Withers, Drew M Brown, Innocent Byiringiro, et al.
The Journal of Biological Chemistry
|
February 25, 2005
Regulation of L-type Ca2+ channel activity and insulin secretion by the Rem2 GTPase
Brian S Finlin, Amber L Mosley, Shawn M Crump, et al.
Journal of the American Heart Association
|
June 25, 2014
Arrhythmogenic calmodulin mutations disrupt intracellular cardiomyocyte Ca2+ regulation by distinct mechanisms
Guo Yin, Faisal Hassan, Ayman R Haroun, et al.
Page
of 6
Search research articles
Search
Showing results (21-30 of 52) with videos related to
Sort By:
Page
of 6
The Journal of Biological Chemistry
|
August 10, 2007
Plasma membrane targeting is essential for Rem-mediated Ca2+ channel inhibition
Robert N Correll, Chunyan Pang, Brian S Finlin, et al.
American Journal of Physiology. Heart and Circulatory Physiology
|
September 16, 2015
Loss of Rad-GTPase produces a novel adaptive cardiac phenotype resistant to systolic decline with aging
Janet R Manning, Catherine N Withers, Bryana Levitan, et al.
The Journal of Biological Chemistry
|
June 23, 2006
Analysis of the complex between Ca2+ channel beta-subunit and the Rem GTPase
Brian S Finlin, Robert N Correll, Chunyan Pang, et al.
Stem Cells and Development
|
May 31, 2008
In vitro electrophysiological drug testing using human embryonic stem cell derived cardiomyocytes
Oren Caspi, Ilanit Itzhaki, Izhak Kehat, et al.
Cell Calcium
|
February 21, 2009
Steady-state coupling of plasma membrane calcium entry to extrusion revealed by novel L-type calcium channel block
William C Lester, Elizabeth A Schroder, Don E Burgess, et al.
Journal of Molecular and Cellular Cardiology
|
February 8, 2021
Rad-GTPase contributes to heart rate via L-type calcium channel regulation
Bryana M Levitan, Brooke M Ahern, Ajoy Aloysius, et al.
Channels (Austin, Tex.)
|
May 12, 2010
Rem GTPase interacts with the proximal CaV1.2 C-terminus and modulates calcium-dependent channel inactivation
Chunyan Pang, Shawn M Crump, Ling Jin, et al.
Bone
|
July 23, 2017
Rad GTPase is essential for the regulation of bone density and bone marrow adipose tissue in mice
Catherine N Withers, Drew M Brown, Innocent Byiringiro, et al.
The Journal of Biological Chemistry
|
February 25, 2005
Regulation of L-type Ca2+ channel activity and insulin secretion by the Rem2 GTPase
Brian S Finlin, Amber L Mosley, Shawn M Crump, et al.
Journal of the American Heart Association
|
June 25, 2014
Arrhythmogenic calmodulin mutations disrupt intracellular cardiomyocyte Ca2+ regulation by distinct mechanisms
Guo Yin, Faisal Hassan, Ayman R Haroun, et al.
Page
of 6