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Jop Kind

Showing results (11-20 of 37) with videos related to

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Methods in Molecular Biology (Clifton, N.J.)|September 18, 2020
DamID to Map Genome-Protein Interactions in Preimplantation Mouse EmbryosMrinmoy Pal, Jop Kind, Maria-Elena Torres-Padilla
Molecular and Cellular Biology|March 19, 2008
Transcription-coupled methylation of histone H3 at lysine 36 regulates dosage compensation by enhancing recruitment of the MSL complex in Drosophila melanogasterOliver Bell, Thomas Conrad, Jop Kind, et al.
NAR Genomics and Bioinformatics|December 31, 2025
A computational framework to dissect imputation strategies for single-cell histone modification dataMarta Moreno-González, Jeroen de Ridder, Jop Kind, et al.
Plos Genetics|February 23, 2010
Nuclear pore proteins nup153 and megator define transcriptionally active regions in the Drosophila genomeJuan M Vaquerizas, Ritsuko Suyama, Jop Kind, et al.
Cell|May 31, 2008
Genome-wide analysis reveals MOF as a key regulator of dosage compensation and gene expression in DrosophilaJop Kind, Juan M Vaquerizas, Philipp Gebhardt, et al.
Genes & Development|December 3, 2014
Differential spatial and structural organization of the X chromosome underlies dosage compensation in C. elegansRahul Sharma, Daniel Jost, Jop Kind, et al.
Cell Reports|June 11, 2013
The genomic landscape of the somatic linker histone subtypes H1.1 to H1.5 in human cellsAnnalisa Izzo, Kinga Kamieniarz-Gdula, Fidel Ramírez, et al.
Immunity|November 20, 2012
The CD27 and CD70 costimulatory pathway inhibits effector function of T helper 17 cells and attenuates associated autoimmunityJonathan M Coquet, Sabine Middendorp, Gerda van der Horst, et al.
Genome Research|November 6, 2012
Constitutive nuclear lamina-genome interactions are highly conserved and associated with A/T-rich sequenceWouter Meuleman, Daan Peric-Hupkes, Jop Kind, et al.
Developmental Cell|March 17, 2012
The MOF chromobarrel domain controls genome-wide H4K16 acetylation and spreading of the MSL complexThomas Conrad, Florence M G Cavalli, Herbert Holz, et al.
Pageof 4

Showing results (11-20 of 37) with videos related to

Sort By:
Pageof 4
Methods in Molecular Biology (Clifton, N.J.)|September 18, 2020
DamID to Map Genome-Protein Interactions in Preimplantation Mouse EmbryosMrinmoy Pal, Jop Kind, Maria-Elena Torres-Padilla
Molecular and Cellular Biology|March 19, 2008
Transcription-coupled methylation of histone H3 at lysine 36 regulates dosage compensation by enhancing recruitment of the MSL complex in Drosophila melanogasterOliver Bell, Thomas Conrad, Jop Kind, et al.
NAR Genomics and Bioinformatics|December 31, 2025
A computational framework to dissect imputation strategies for single-cell histone modification dataMarta Moreno-González, Jeroen de Ridder, Jop Kind, et al.
Plos Genetics|February 23, 2010
Nuclear pore proteins nup153 and megator define transcriptionally active regions in the Drosophila genomeJuan M Vaquerizas, Ritsuko Suyama, Jop Kind, et al.
Cell|May 31, 2008
Genome-wide analysis reveals MOF as a key regulator of dosage compensation and gene expression in DrosophilaJop Kind, Juan M Vaquerizas, Philipp Gebhardt, et al.
Genes & Development|December 3, 2014
Differential spatial and structural organization of the X chromosome underlies dosage compensation in C. elegansRahul Sharma, Daniel Jost, Jop Kind, et al.
Cell Reports|June 11, 2013
The genomic landscape of the somatic linker histone subtypes H1.1 to H1.5 in human cellsAnnalisa Izzo, Kinga Kamieniarz-Gdula, Fidel Ramírez, et al.
Immunity|November 20, 2012
The CD27 and CD70 costimulatory pathway inhibits effector function of T helper 17 cells and attenuates associated autoimmunityJonathan M Coquet, Sabine Middendorp, Gerda van der Horst, et al.
Genome Research|November 6, 2012
Constitutive nuclear lamina-genome interactions are highly conserved and associated with A/T-rich sequenceWouter Meuleman, Daan Peric-Hupkes, Jop Kind, et al.
Developmental Cell|March 17, 2012
The MOF chromobarrel domain controls genome-wide H4K16 acetylation and spreading of the MSL complexThomas Conrad, Florence M G Cavalli, Herbert Holz, et al.
Pageof 4