Showing results (61-70 of 98) with videos related to

Sort By:
Pageof 10
BMC Neuroscience|November 30, 2007
Dynamic and redundant regulation of LRRK2 and LRRK1 expressionSaskia Biskup, Darren J Moore, Alexis Rea, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 8, 2020
Dopaminergic neurodegeneration induced by Parkinson's disease-linked G2019S LRRK2 is dependent on kinase and GTPase activityAn Phu Tran Nguyen, Elpida Tsika, Kaela Kelly, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 5, 2005
Parkinson's disease-associated mutations in leucine-rich repeat kinase 2 augment kinase activityAndrew B West, Darren J Moore, Saskia Biskup, et al.
Brain Communications|October 27, 2021
Neuronal VPS35 deletion induces spinal cord motor neuron degeneration and early post-natal lethalityDorian Sargent, Lindsey A Cunningham, Dylan J Dues, et al.
Translational Neurodegeneration|June 19, 2025
Pathological α-synuclein elicits granulovacuolar degeneration independent of tauDylan J Dues, Madalynn L Erb, Alysa Kasen, et al.
Plos Genetics|April 14, 2010
GTPase activity plays a key role in the pathobiology of LRRK2Yulan Xiong, Candice E Coombes, Austin Kilaru, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 15, 2005
Leucine-rich repeat kinase 2 (LRRK2) interacts with parkin, and mutant LRRK2 induces neuronal degenerationWanli W Smith, Zhong Pei, Haibing Jiang, et al.
NPJ Parkinson'S Disease|July 8, 2026
Interaction of G2019S LRRK2 and metabolic syndrome in a two-hit mouse model of Parkinson's disease: LRRK2-driven systemic depletion of pyrimidine nucleosidesYue Ma, Madalynn L Erb, Kayla A Sipple, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 8, 2019
Parkinson's disease-linked D620N VPS35 knockin mice manifest tau neuropathology and dopaminergic neurodegenerationXi Chen, Jennifer K Kordich, Erin T Williams, et al.
Scientific Reports|June 3, 2017
GLP-1 receptor signalling promotes β-cell glucose metabolism via mTOR-dependent HIF-1α activationRodrigo Carlessi, Younan Chen, Jordan Rowlands, et al.
Pageof 10