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Open Biology|September 3, 2020
Neutrophil interactions with the sexually transmitted parasite <i>Trichomonas vaginalis:</i> implications for immunity and pathogenesisSuhani B Bhakta, Jose A Moran, Frances MercerFrontiers in Immunology|June 13, 2022
Contribution of Sex Differences to HIV Immunology, Pathogenesis, and Cure ApproachesJose A Moran, Shireen R Turner, Matthew D MarsdenStem Cells and Development|March 11, 2021
Downregulation of SUV39H1 and CITED2 Exerts Additive Effect on Promoting Adipogenic Commitment of Human Mesenchymal Stem CellsLun Tan, Linh Tran, Stephanie Ferreyra, et al.Virology|February 26, 2023
Secreted factors induced by PKC modulators do not indirectly cause HIV latency reversalJose A Moran, Alok Ranjan, Rami Hourani, et al.Frontiers in Physiology|August 1, 2018
Nanotherapeutics for Treatment of Pulmonary Arterial HypertensionVictor Segura-Ibarra, Suhong Wu, Nida Hassan, et al.Parasite Immunology|February 19, 2024
Complement receptor 3 is required for maximum in vitro trogocytic killing of the parasite Trichomonas vaginalis by human neutrophil-like cellsEmma N Trujillo, Barbara A Flores, Isabel V Romero, et al.The American Surgeon|May 17, 2024
Epiploic Appendagitis: Systematic Review of a Distinctive PathologyCarlos D Acevedo-Castillo, Hannia M Macias-Cruz, Arantxa Ramirez-Cisneros, et al.Science Advances|January 24, 2025
Synthesis and preclinical evaluation of tigilanol tiglate analogs as latency-reversing agents for the eradication of HIVZachary O Gentry, Owen D McAteer, Jennifer L Hamad, et al.Pathogens & Immunity|May 20, 2024
Defining the Effects of PKC Modulator HIV Latency-Reversing Agents on Natural Killer CellsMelanie Dimapasoc, Jose A Moran, Steve W Cole, et al.Pageof 2