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Pflugers Archiv : European Journal of Physiology|November 19, 2009
K(ATP) channelopathies in the pancreasMaria S Remedi, Joseph C KosterDiabetes|October 27, 2005
Diabetes and insulin secretion: the ATP-sensitive K+ channel (K ATP) connectionJoseph C Koster, M Alan Permutt, Colin G NicholsBiophysical Journal|August 19, 2008
DEND mutation in Kir6.2 (KCNJ11) reveals a flexible N-terminal region critical for ATP-sensing of the KATP channelJoseph C Koster, Harley T Kurata, Decha Enkvetchakul, et al.Diabetes|August 27, 2005
ATP and sulfonylurea sensitivity of mutant ATP-sensitive K+ channels in neonatal diabetes: implications for pharmacogenomic therapyJoseph C Koster, Maria S Remedi, Crystal Dao, et al.The Journal of Biological Chemistry|September 25, 2010
Effects of the HIV protease inhibitor ritonavir on GLUT4 knock-out miceArpita Kalla Vyas, Joseph C Koster, Anatoly Tzekov, et al.Physiological Reviews|July 29, 2010
Muscle KATP channels: recent insights to energy sensing and myoprotectionThomas P Flagg, Decha Enkvetchakul, Joseph C Koster, et al.Diabetes|September 28, 2005
ATP-sensitive K+ channel signaling in glucokinase-deficient diabetesMaria S Remedi, Joseph C Koster, Brian L Patton, et al.The Journal of Clinical Endocrinology and Metabolism|December 13, 2007
The G53D mutation in Kir6.2 (KCNJ11) is associated with neonatal diabetes and motor dysfunction in adulthood that is improved with sulfonylurea therapyJoseph C Koster, Francesco Cadario, Cinzia Peruzzi, et al.Diabetes|June 28, 2003
HIV protease inhibitors acutely impair glucose-stimulated insulin releaseJoseph C Koster, Maria S Remedi, Haijun Qiu, et al.Diabetes|October 27, 2006
Expression of ATP-insensitive KATP channels in pancreatic beta-cells underlies a spectrum of diabetic phenotypesJoseph C Koster, Maria S Remedi, Ricard Masia, et al.Pageof 2