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Neurobiology of Disease|December 20, 2011
A new mouse model for the slow-channel congenital myasthenic syndrome induced by the AChR εL221F mutationFrédéric Chevessier, Christoph Peter, Ulrike Mersdorf, et al.
The Journal of Pharmacology and Experimental Therapeutics|August 6, 2009
Capsaicin protects mouse neuromuscular junctions from the neuroparalytic effects of botulinum neurotoxin aBaskaran Thyagarajan, Natalia Krivitskaya, Joseph G Potian, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|December 16, 2006
Glucose, insulin, and leptin signaling pathways modulate nitric oxide synthesis in glucose-inhibited neurons in the ventromedial hypothalamusDebra D Canabal, Zhentao Song, Joseph G Potian, et al.
The Journal of Biological Chemistry|November 29, 2001
Identification of residues at the alpha and epsilon subunit interfaces mediating species selectivity of Waglerin-1 for nicotinic acetylcholine receptorsBrian E Molles, Parastoo Rezai, Eric F Kline, et al.
Frontiers in Human Neuroscience|July 13, 2013
Spatial and temporal features of superordinate semantic processing studied with fMRI and EEGMichelle E Costanzo, Joseph J McArdle, Bruce Swett, et al.
American Journal of Physiology. Endocrinology and Metabolism|July 16, 2009
Motor unit number estimate as a predictor of motor dysfunction in an animal model of type 1 diabetesNizar Souayah, Joseph G Potian, Carmen C Garcia, et al.
Plos One|December 2, 2014
Altered active zones, vesicle pools, nerve terminal conductivity, and morphology during experimental MuSK myasthenia gravisVishwendra Patel, Anne Oh, Antanina Voit, et al.
Developmental Neurobiology|April 18, 2008
The rescue of developing avian motoneurons from programmed cell death by a selective inhibitor of the fetal muscle-specific nicotinic acetylcholine receptorRonald W Oppenheim, Jordi Calderó, Doloros Cuitat, et al.
Annals of the New York Academy of Sciences|June 24, 2008
Peptide-toxin tools for probing the expression and function of fetal and adult subtypes of the nicotinic acetylcholine receptorRussell W Teichert, Carmen C Garcia, Joseph G Potian, et al.
American Journal of Physiology. Endocrinology and Metabolism|June 29, 2012
Acetylcholinesterase deficiency contributes to neuromuscular junction dysfunction in type 1 diabetic neuropathyCarmen C Garcia, Joseph G Potian, Kormakur Hognason, et al.
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