Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Joseph T Coyle

Showing results (121-130 of 131) with videos related to

Pageof 14
Sort By:
Molecular Psychiatry|August 2, 2019
Correction: iPSC-derived homogeneous populations of developing schizophrenia cortical interneurons have compromised mitochondrial functionPeiyan Ni, Haneul Noh, Gun-Hoo Park, et al.
Biorxiv : the Preprint Server for Biology|July 3, 2023
A marker chromosome in psychosis identifies glycine decarboxylase (GLDC) as a novel regulator of neuronal and synaptic function in the hippocampusMaltesh Kambali, Yan Li, Petr Unichenko, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 31, 2021
Clinical evidence that a dysregulated master neural network modulator may aid in diagnosing schizophreniaMunetaka Nomoto, Glenn T Konopaske, Naoya Yamashita, et al.
Molecular Psychiatry|June 14, 2022
Impact of schizophrenia GWAS loci converge onto distinct pathways in cortical interneurons vs glutamatergic neurons during developmentDongxin Liu, Amy Zinski, Akanksha Mishra, et al.
Nature Neuroscience|October 24, 2020
Activated microglia cause metabolic disruptions in developmental cortical interneurons that persist in interneurons from individuals with schizophreniaGun-Hoo Park, Haneul Noh, Zhicheng Shao, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 4, 2021
Dopaminergic neuromodulation of prefrontal cortex activity requires the NMDA receptor coagonist d-serineGlenn Dallérac, Xia Li, Pierre Lecouflet, et al.
Molecular Psychiatry|August 29, 2024
An increased copy number of glycine decarboxylase (GLDC) associated with psychosis reduces extracellular glycine and impairs NMDA receptor functionMaltesh Kambali, Yan Li, Petr Unichenko, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|August 30, 2008
The role of animal models in evaluating reasonable safety and efficacy for human trials of cell-based interventions for neurologic conditionsAlan Regenberg, Debra J H Mathews, David M Blass, et al.
Biological Psychiatry|July 8, 2019
Targeted Treatment of Individuals With Psychosis Carrying a Copy Number Variant Containing a Genomic Triplication of the Glycine Decarboxylase GeneJ Alexander Bodkin, Michael J Coleman, Laura J Godfrey, et al.
Nature Neuroscience|January 22, 2019
Dysregulated protocadherin-pathway activity as an intrinsic defect in induced pluripotent stem cell-derived cortical interneurons from subjects with schizophreniaZhicheng Shao, Haneul Noh, Woong Bin Kim, et al.
Pageof 14

Showing results (121-130 of 131) with videos related to

Sort By:
Pageof 14
Molecular Psychiatry|August 2, 2019
Correction: iPSC-derived homogeneous populations of developing schizophrenia cortical interneurons have compromised mitochondrial functionPeiyan Ni, Haneul Noh, Gun-Hoo Park, et al.
Biorxiv : the Preprint Server for Biology|July 3, 2023
A marker chromosome in psychosis identifies glycine decarboxylase (GLDC) as a novel regulator of neuronal and synaptic function in the hippocampusMaltesh Kambali, Yan Li, Petr Unichenko, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 31, 2021
Clinical evidence that a dysregulated master neural network modulator may aid in diagnosing schizophreniaMunetaka Nomoto, Glenn T Konopaske, Naoya Yamashita, et al.
Molecular Psychiatry|June 14, 2022
Impact of schizophrenia GWAS loci converge onto distinct pathways in cortical interneurons vs glutamatergic neurons during developmentDongxin Liu, Amy Zinski, Akanksha Mishra, et al.
Nature Neuroscience|October 24, 2020
Activated microglia cause metabolic disruptions in developmental cortical interneurons that persist in interneurons from individuals with schizophreniaGun-Hoo Park, Haneul Noh, Zhicheng Shao, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 4, 2021
Dopaminergic neuromodulation of prefrontal cortex activity requires the NMDA receptor coagonist d-serineGlenn Dallérac, Xia Li, Pierre Lecouflet, et al.
Molecular Psychiatry|August 29, 2024
An increased copy number of glycine decarboxylase (GLDC) associated with psychosis reduces extracellular glycine and impairs NMDA receptor functionMaltesh Kambali, Yan Li, Petr Unichenko, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|August 30, 2008
The role of animal models in evaluating reasonable safety and efficacy for human trials of cell-based interventions for neurologic conditionsAlan Regenberg, Debra J H Mathews, David M Blass, et al.
Biological Psychiatry|July 8, 2019
Targeted Treatment of Individuals With Psychosis Carrying a Copy Number Variant Containing a Genomic Triplication of the Glycine Decarboxylase GeneJ Alexander Bodkin, Michael J Coleman, Laura J Godfrey, et al.
Nature Neuroscience|January 22, 2019
Dysregulated protocadherin-pathway activity as an intrinsic defect in induced pluripotent stem cell-derived cortical interneurons from subjects with schizophreniaZhicheng Shao, Haneul Noh, Woong Bin Kim, et al.
Pageof 14