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Translational Psychiatry|December 15, 2018
Dopamine perturbation of gene co-expression networks reveals differential response in schizophrenia for translational machineryMark Z Kos, Jubao Duan, Alan R Sanders, et al.Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|May 19, 2021
Sex-specific nicotine sensitization and imprinting of self-administration in rats inform GWAS findings on human addiction phenotypesAlena Kozlova, Robert R Butler, Siwei Zhang, et al.Human Molecular Genetics|May 30, 2015
Transcriptome outlier analysis implicates schizophrenia susceptibility genes and enriches putatively functional rare genetic variantsJubao Duan, Alan R Sanders, Winton Moy, et al.Science Advances|September 20, 2019
Deconvolution of transcriptional networks identifies TCF4 as a master regulator in schizophreniaAbolfazl Doostparast Torshizi, Chris Armoskus, Hanwen Zhang, et al.Plos Genetics|October 23, 2024
A comprehensive framework for trans-ancestry pathway analysis using GWAS summary data from diverse populationsSheng Fu, William Wheeler, Xiaoyu Wang, et al.Molecular Neuropsychiatry|May 14, 2020
The Genetic Relevance of Human Induced Pluripotent Stem Cell-Derived Microglia to Alzheimer's Disease and Major Neuropsychiatric DisordersRobert R Butler Iii, Alena Kozlova, Hanwen Zhang, et al.Journal of Neuroscience Methods|February 18, 2020
ASCL1- and DLX2-induced GABAergic neurons from hiPSC-derived NPCsNatalie Barretto, Hanwen Zhang, Samuel K Powell, et al.American Journal of Human Genetics|August 27, 2004
Polymorphisms in the trace amine receptor 4 (TRAR4) gene on chromosome 6q23.2 are associated with susceptibility to schizophreniaJubao Duan, Maria Martinez, Alan R Sanders, et al.Proceedings of the National Academy of Sciences of the United States of America|July 13, 2016
Reversal of dendritic phenotypes in 16p11.2 microduplication mouse model neurons by pharmacological targeting of a network hubKatherine D Blizinsky, Blanca Diaz-Castro, Marc P Forrest, et al.Nature Communications|September 17, 2014
A sequence variant in human KALRN impairs protein function and coincides with reduced cortical thicknessTheron A Russell, Katherine D Blizinsky, Derin J Cobia, et al.Pageof 9