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JAK-STAT|September 24, 2013
The JAK-STAT pathway and hematopoietic stem cells from the JAK2 V617F perspectiveJudith Staerk, Stefan N Constantinescu
The Journal of Biological Chemistry|October 22, 2005
JAK1 and Tyk2 activation by the homologous polycythemia vera JAK2 V617F mutation: cross-talk with IGF1 receptorJudith Staerk, Anders Kallin, Jean-Baptiste Demoulin, et al.
The Journal of Biological Chemistry|May 19, 2005
Janus kinases affect thrombopoietin receptor cell surface localization and stabilityYohan Royer, Judith Staerk, Marcel Costuleanu, et al.
Journal of Cellular and Molecular Medicine|March 9, 2018
Cytokinesis arrest and multiple centrosomes in B cell chronic lymphocytic leukaemiaMarie Rogne, Oksana Svaerd, Julia Madsen-Østerbye, et al.
Stem Cells (Dayton, Ohio)|July 12, 2020
DNMT3B deficiency alters mitochondrial biogenesis and α-ketoglutarate levels in human embryonic stem cellsArtur Cieslar-Pobuda, Theresa D Ahrens, Safak Caglayan, et al.
The Journal of Biological Chemistry|March 31, 2019
The kinase PERK and the transcription factor ATF4 play distinct and essential roles in autophagy resulting from tunicamycin-induced ER stressMorten Luhr, Maria Lyngaas Torgersen, Paula Szalai, et al.
The Journal of Biological Chemistry|March 8, 2008
Substitution of pseudokinase domain residue Val-617 by large non-polar amino acids causes activation of JAK2Alexandra Dusa, Judith Staerk, Joanne Elliott, et al.
The Journal of Biological Chemistry|January 14, 2009
Acute lymphoblastic leukemia-associated JAK1 mutants activate the Janus kinase/STAT pathway via interleukin-9 receptor alpha homodimersTekla Hornakova, Judith Staerk, Yohan Royer, et al.
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