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Medrxiv : the Preprint Server for Health Sciences|February 23, 2026
Wild-type C9orf72 expression is a genetic modifier of C9-ALS survivalStanislav Tsitkov, Akshay Raju, Jie Wu, et al.Neuron|July 6, 2019
Small-Molecule Modulation of TDP-43 Recruitment to Stress Granules Prevents Persistent TDP-43 Accumulation in ALS/FTDMark Y Fang, Sebastian Markmiller, Anthony Q Vu, et al.Proceedings of the National Academy of Sciences of the United States of America|February 13, 2013
Astrocyte pathology and the absence of non-cell autonomy in an induced pluripotent stem cell model of TDP-43 proteinopathyAndrea Serio, Bilada Bilican, Sami J Barmada, et al.Proceedings of the National Academy of Sciences of the United States of America|November 12, 2014
Potential function for the Huntingtin protein as a scaffold for selective autophagyJoseph Ochaba, Tamás Lukacsovich, George Csikos, et al.Plos Pathogens|January 5, 2023
Nuclear accumulation of host transcripts during Zika Virus InfectionKristoffer E Leon, Mir M Khalid, Ryan A Flynn, et al.Iscience|November 8, 2021
An integrated multi-omic analysis of iPSC-derived motor neurons from C9ORF72 ALS patients, Jonathan Li, Ryan G Lim, et al.The Journal of Clinical Investigation|February 11, 2014
ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defectsHaiyan Qiu, Sebum Lee, Yulei Shang, et al.Proceedings of the National Academy of Sciences of the United States of America|July 23, 2013
Targeting H3K4 trimethylation in Huntington diseaseMalini Vashishtha, Christopher W Ng, Ferah Yildirim, et al.Proceedings of the National Academy of Sciences of the United States of America|March 28, 2012
Mutant induced pluripotent stem cell lines recapitulate aspects of TDP-43 proteinopathies and reveal cell-specific vulnerabilityBilada Bilican, Andrea Serio, Sami J Barmada, et al.Science Translational Medicine|June 7, 2013
Comment on "Drug screening for ALS using patient-specific induced pluripotent stem cells"Bilada Bilican, Andrea Serio, Sami J Barmada, et al.Pageof 14