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Bone
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November 4, 2017
Variable signaling activity by FOP ACVR1 mutations
Julia Haupt, Meiqi Xu, Eileen M Shore
Gene Expression Patterns : GEP
|
March 24, 2006
Comparative expression pattern of Odd-skipped related genes Osr1 and Osr2 in chick embryonic development
Sigmar Stricker, Norbert Brieske, Julia Haupt, et al.
Cold Spring Harbor Perspectives in Biology
|
March 2, 2017
TGF-β Family Signaling in Connective Tissue and Skeletal Diseases
Elena Gallo MacFarlane, Julia Haupt, Harry C Dietz, et al.
Stem Cells and Development
|
June 16, 2011
Odd-skipped related genes regulate differentiation of embryonic limb mesenchyme and bone marrow mesenchymal stromal cells
Sigmar Stricker, Susanne Mathia, Julia Haupt, et al.
Stem Cell Reviews and Reports
|
January 7, 2021
ActivinA Induced SMAD1/5 Signaling in an iPSC Derived EC Model of Fibrodysplasia Ossificans Progressiva (FOP) Can Be Rescued by the Drug Candidate Saracatinib
Susanne Hildebrandt, Branka Kampfrath, Kristin Fischer, et al.
Methods in Enzymology
|
November 2, 2010
Investigations of activated ACVR1/ALK2, a bone morphogenetic protein type I receptor, that causes fibrodysplasia ossificans progressiva
Frederick S Kaplan, Petra Seemann, Julia Haupt, et al.
The Journal of Clinical Investigation
|
May 12, 2010
Homeobox genes d11-d13 and a13 control mouse autopod cortical bone and joint formation
Pablo Villavicencio-Lorini, Pia Kuss, Julia Friedrich, et al.
Molecular Biology of the Cell
|
November 1, 2018
ACVR1<sup>R206H</sup> FOP mutation alters mechanosensing and tissue stiffness during heterotopic ossification
Julia Haupt, Alexandra Stanley, Claire M McLeod, et al.
The Journal of Clinical Investigation
|
October 27, 2009
The fibrodysplasia ossificans progressiva R206H ACVR1 mutation activates BMP-independent chondrogenesis and zebrafish embryo ventralization
Qi Shen, Shawn C Little, Meiqi Xu, et al.
Human Molecular Genetics
|
May 24, 2014
ACVR1 p.Q207E causes classic fibrodysplasia ossificans progressiva and is functionally distinct from the engineered constitutively active ACVR1 p.Q207D variant
Julia Haupt, Alexandra Deichsel, Katja Stange, et al.
Page
of 2
Search research articles
Search
Showing results (1-10 of 11) with videos related to
Sort By:
Page
of 2
Bone
|
November 4, 2017
Variable signaling activity by FOP ACVR1 mutations
Julia Haupt, Meiqi Xu, Eileen M Shore
Gene Expression Patterns : GEP
|
March 24, 2006
Comparative expression pattern of Odd-skipped related genes Osr1 and Osr2 in chick embryonic development
Sigmar Stricker, Norbert Brieske, Julia Haupt, et al.
Cold Spring Harbor Perspectives in Biology
|
March 2, 2017
TGF-β Family Signaling in Connective Tissue and Skeletal Diseases
Elena Gallo MacFarlane, Julia Haupt, Harry C Dietz, et al.
Stem Cells and Development
|
June 16, 2011
Odd-skipped related genes regulate differentiation of embryonic limb mesenchyme and bone marrow mesenchymal stromal cells
Sigmar Stricker, Susanne Mathia, Julia Haupt, et al.
Stem Cell Reviews and Reports
|
January 7, 2021
ActivinA Induced SMAD1/5 Signaling in an iPSC Derived EC Model of Fibrodysplasia Ossificans Progressiva (FOP) Can Be Rescued by the Drug Candidate Saracatinib
Susanne Hildebrandt, Branka Kampfrath, Kristin Fischer, et al.
Methods in Enzymology
|
November 2, 2010
Investigations of activated ACVR1/ALK2, a bone morphogenetic protein type I receptor, that causes fibrodysplasia ossificans progressiva
Frederick S Kaplan, Petra Seemann, Julia Haupt, et al.
The Journal of Clinical Investigation
|
May 12, 2010
Homeobox genes d11-d13 and a13 control mouse autopod cortical bone and joint formation
Pablo Villavicencio-Lorini, Pia Kuss, Julia Friedrich, et al.
Molecular Biology of the Cell
|
November 1, 2018
ACVR1<sup>R206H</sup> FOP mutation alters mechanosensing and tissue stiffness during heterotopic ossification
Julia Haupt, Alexandra Stanley, Claire M McLeod, et al.
The Journal of Clinical Investigation
|
October 27, 2009
The fibrodysplasia ossificans progressiva R206H ACVR1 mutation activates BMP-independent chondrogenesis and zebrafish embryo ventralization
Qi Shen, Shawn C Little, Meiqi Xu, et al.
Human Molecular Genetics
|
May 24, 2014
ACVR1 p.Q207E causes classic fibrodysplasia ossificans progressiva and is functionally distinct from the engineered constitutively active ACVR1 p.Q207D variant
Julia Haupt, Alexandra Deichsel, Katja Stange, et al.
Page
of 2