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Julia Ritterhoff

Showing results (11-20 of 24) with videos related to

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Circulation|November 15, 2023
Raising NAD<sup>+</sup> Level Stimulates Short-Chain Dehydrogenase/Reductase Proteins to Alleviate Heart Failure Independent of Mitochondrial Protein DeacetylationMatthew A Walker, Hongye Chen, Aprajita Yadav, et al.
Circulation Research|November 12, 2019
Metabolic Remodeling Promotes Cardiac Hypertrophy by Directing Glucose to Aspartate BiosynthesisJulia Ritterhoff, Sara Young, Outi Villet, et al.
Shock (Augusta, Ga.)|April 8, 2026
A Meta-Analytic Perspective on Echocardiographic Signs of Long-Term Cardiac Dysfunction After Burns, Sepsis, and TraumaTobias Niederegger, Emre Karakas, Thomas Schaschinger, et al.
Nature Communications|July 28, 2018
Glucose promotes cell growth by suppressing branched-chain amino acid degradationDan Shao, Outi Villet, Zhen Zhang, et al.
Burns : Journal of the International Society for Burn Injuries|November 26, 2025
The burn repair molecule? Evaluating FGF-21 in thermal injury - A systematic reviewTobias Niederegger, Thomas Schaschinger, Jule Brandt, et al.
Cell Metabolism|February 9, 2017
Defective Branched-Chain Amino Acid Catabolism Disrupts Glucose Metabolism and Sensitizes the Heart to Ischemia-Reperfusion InjuryTao Li, Zhen Zhang, Stephen C Kolwicz, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|May 26, 2015
S100A1 DNA-based Inotropic Therapy Protects Against Proarrhythmogenic Ryanodine Receptor 2 DysfunctionJulia Ritterhoff, Mirko Völkers, Andreas Seitz, et al.
Circulation Research|October 11, 2012
S100A1 deficiency impairs postischemic angiogenesis via compromised proangiogenic endothelial cell function and nitric oxide synthase regulationPatrick Most, Carolin Lerchenmüller, Giuseppe Rengo, et al.
The Journal of Clinical Investigation|May 16, 2022
Upregulation of mitochondrial ATPase inhibitory factor 1 (ATPIF1) mediates increased glycolysis in mouse heartsBo Zhou, Arianne Caudal, Xiaoting Tang, et al.
Circulation. Heart Failure|June 19, 2025
Cardiac-Targeted AAV5-S100A1 Gene Therapy Protects Against Adverse Remodeling and Contractile Dysfunction in Postischemic HeartsDorothea Kehr, Janek Salatzki, Birgit Seger, et al.
Pageof 3

Showing results (11-20 of 24) with videos related to

Sort By:
Pageof 3
Circulation|November 15, 2023
Raising NAD<sup>+</sup> Level Stimulates Short-Chain Dehydrogenase/Reductase Proteins to Alleviate Heart Failure Independent of Mitochondrial Protein DeacetylationMatthew A Walker, Hongye Chen, Aprajita Yadav, et al.
Circulation Research|November 12, 2019
Metabolic Remodeling Promotes Cardiac Hypertrophy by Directing Glucose to Aspartate BiosynthesisJulia Ritterhoff, Sara Young, Outi Villet, et al.
Shock (Augusta, Ga.)|April 8, 2026
A Meta-Analytic Perspective on Echocardiographic Signs of Long-Term Cardiac Dysfunction After Burns, Sepsis, and TraumaTobias Niederegger, Emre Karakas, Thomas Schaschinger, et al.
Nature Communications|July 28, 2018
Glucose promotes cell growth by suppressing branched-chain amino acid degradationDan Shao, Outi Villet, Zhen Zhang, et al.
Burns : Journal of the International Society for Burn Injuries|November 26, 2025
The burn repair molecule? Evaluating FGF-21 in thermal injury - A systematic reviewTobias Niederegger, Thomas Schaschinger, Jule Brandt, et al.
Cell Metabolism|February 9, 2017
Defective Branched-Chain Amino Acid Catabolism Disrupts Glucose Metabolism and Sensitizes the Heart to Ischemia-Reperfusion InjuryTao Li, Zhen Zhang, Stephen C Kolwicz, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|May 26, 2015
S100A1 DNA-based Inotropic Therapy Protects Against Proarrhythmogenic Ryanodine Receptor 2 DysfunctionJulia Ritterhoff, Mirko Völkers, Andreas Seitz, et al.
Circulation Research|October 11, 2012
S100A1 deficiency impairs postischemic angiogenesis via compromised proangiogenic endothelial cell function and nitric oxide synthase regulationPatrick Most, Carolin Lerchenmüller, Giuseppe Rengo, et al.
The Journal of Clinical Investigation|May 16, 2022
Upregulation of mitochondrial ATPase inhibitory factor 1 (ATPIF1) mediates increased glycolysis in mouse heartsBo Zhou, Arianne Caudal, Xiaoting Tang, et al.
Circulation. Heart Failure|June 19, 2025
Cardiac-Targeted AAV5-S100A1 Gene Therapy Protects Against Adverse Remodeling and Contractile Dysfunction in Postischemic HeartsDorothea Kehr, Janek Salatzki, Birgit Seger, et al.
Pageof 3