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Plos One|May 12, 2011
CEACAM6 gene variants in inflammatory bowel diseaseJürgen Glas, Julia Seiderer, Christoph Fries, et al.
The American Journal of Gastroenterology|March 4, 2010
Disease activity, ANCA, and IL23R genotype status determine early response to infliximab in patients with ulcerative colitisMatthias Jürgens, Rüdiger P Laubender, Franziska Hartl, et al.
Journal of Gastroenterology|April 30, 2010
The presence of fistulas and NOD2 homozygosity strongly predict intestinal stenosis in Crohn's disease independent of the IL23R genotypeMatthias Jürgens, Stephan Brand, Rüdiger P Laubender, et al.
Inflammatory Bowel Diseases|June 29, 2006
Role of the NFKB1 -94ins/delATTG promoter polymorphism in IBD and potential interactions with polymorphisms in the CARD15/NOD2, IKBL, and IL-1RN genesJürgen Glas, Helga-Paula Török, Laurian Tonenchi, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|March 16, 2006
IL-22 is increased in active Crohn's disease and promotes proinflammatory gene expression and intestinal epithelial cell migrationStephan Brand, Florian Beigel, Torsten Olszak, et al.
Wiener Klinische Wochenschrift|October 19, 2007
A systematic survey evaluating 6-thioguanine-related hepatotoxicity in patients with inflammatory bowel diseaseAlexander Teml, Matthias Schwab, Daan W Hommes, et al.
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