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Journal of Neuroimmunology|May 6, 2014
Apolipoprotein E mediation of neuro-inflammation in a murine model of multiple sclerosisSoomin Shin, Katharine A Walz, Angela S Archambault, et al.Plos One|November 9, 2011
Batf3-dependent CD11b(low/-) peripheral dendritic cells are GM-CSF-independent and are not required for Th cell priming after subcutaneous immunizationBrian T Edelson, Tara R Bradstreet, Wumesh KC, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 22, 2015
B cell antigen presentation is sufficient to drive neuroinflammation in an animal model of multiple sclerosisChelsea R Parker Harp, Angela S Archambault, Julia Sim, et al.Nature Communications|April 5, 2014
Bhlhe40 controls cytokine production by T cells and is essential for pathogenicity in autoimmune neuroinflammationChih-Chung Lin, Tara R Bradstreet, Elizabeth A Schwarzkopf, et al.Journal of Immunology (Baltimore, Md. : 1950)|June 18, 2013
Cutting edge: Conditional MHC class II expression reveals a limited role for B cell antigen presentation in primary and secondary CD4 T cell responsesAngela S Archambault, Javier A Carrero, Lisa G Barnett, et al.Nature Immunology|June 11, 2003
BTLA is a lymphocyte inhibitory receptor with similarities to CTLA-4 and PD-1Norihiko Watanabe, Maya Gavrieli, John R Sedy, et al.Blood|July 17, 2010
ITAM signaling in dendritic cells controls T helper cell priming by regulating MHC class II recyclingDaniel B Graham, Holly M Akilesh, Grzegorz B Gmyrek, et al.The Journal of Experimental Medicine|February 3, 2016
IL-1-induced Bhlhe40 identifies pathogenic T helper cells in a model of autoimmune neuroinflammationChih-Chung Lin, Tara R Bradstreet, Elizabeth A Schwarzkopf, et al.Nature|July 7, 2009
The AP-1 transcription factor Batf controls T(H)17 differentiationBarbara U Schraml, Kai Hildner, Wataru Ise, et al.Pageof 2