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Cell Transplantation
|
February 5, 2005
The potential of bone marrow stem cells to correct liver dysfunction in a mouse model of Wilson's disease
Katrina J Allen, Daphne M Y Cheah, Xiao Ling Lee, et al.
Physiological Reports
|
April 19, 2014
Effects of ATP7A overexpression in mice on copper transport and metabolism in lactation and gestation
Jarrod Wadwa, Yu-Hsiang Chu, Nhu Nguyen, et al.
Neurobiology of Aging
|
November 2, 2013
Copper pathology in vulnerable brain regions in Parkinson's disease
Katherine M Davies, Sylvain Bohic, Asunción Carmona, et al.
American Journal of Physiology. Cell Physiology
|
June 27, 2008
Copper is taken up efficiently from albumin and alpha2-macroglobulin by cultured human cells by more than one mechanism
Mizue Moriya, Yi-Hsuan Ho, Anne Grana, et al.
The Journal of Biological Chemistry
|
June 23, 2010
Role of glutaredoxin1 and glutathione in regulating the activity of the copper-transporting P-type ATPases, ATP7A and ATP7B
William C J Singleton, Kelly T McInnes, Michael A Cater, et al.
Cancer Research
|
May 19, 2007
Copper-transporting P-type ATPase, ATP7A, confers multidrug resistance and its expression is related to resistance to SN-38 in clinical colon cancer
Satsuki Owatari, Satoshi Akune, Masaharu Komatsu, et al.
Metallomics : Integrated Biometal Science
|
June 14, 2016
Characterizing the molecular phenotype of an Atp7a(T985I) conditional knock in mouse model for X-linked distal hereditary motor neuropathy (dHMNX)
Gonzalo Perez-Siles, Adrienne Grant, Melina Ellis, et al.
American Journal of Human Genetics
|
February 23, 2010
Missense mutations in the copper transporter gene ATP7A cause X-linked distal hereditary motor neuropathy
Marina L Kennerson, Garth A Nicholson, Stephen G Kaler, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 6, 2014
Oral treatment with Cu(II)(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis
Blaine R Roberts, Nastasia K H Lim, Erin J McAllum, et al.
Page
of 5
Search research articles
Search
Showing results (41-50 of 49) with videos related to
Sort By:
Page
of 5
You have reached the last page of results.
This site can display upto 49 results.
Cell Transplantation
|
February 5, 2005
The potential of bone marrow stem cells to correct liver dysfunction in a mouse model of Wilson's disease
Katrina J Allen, Daphne M Y Cheah, Xiao Ling Lee, et al.
Physiological Reports
|
April 19, 2014
Effects of ATP7A overexpression in mice on copper transport and metabolism in lactation and gestation
Jarrod Wadwa, Yu-Hsiang Chu, Nhu Nguyen, et al.
Neurobiology of Aging
|
November 2, 2013
Copper pathology in vulnerable brain regions in Parkinson's disease
Katherine M Davies, Sylvain Bohic, Asunción Carmona, et al.
American Journal of Physiology. Cell Physiology
|
June 27, 2008
Copper is taken up efficiently from albumin and alpha2-macroglobulin by cultured human cells by more than one mechanism
Mizue Moriya, Yi-Hsuan Ho, Anne Grana, et al.
The Journal of Biological Chemistry
|
June 23, 2010
Role of glutaredoxin1 and glutathione in regulating the activity of the copper-transporting P-type ATPases, ATP7A and ATP7B
William C J Singleton, Kelly T McInnes, Michael A Cater, et al.
Cancer Research
|
May 19, 2007
Copper-transporting P-type ATPase, ATP7A, confers multidrug resistance and its expression is related to resistance to SN-38 in clinical colon cancer
Satsuki Owatari, Satoshi Akune, Masaharu Komatsu, et al.
Metallomics : Integrated Biometal Science
|
June 14, 2016
Characterizing the molecular phenotype of an Atp7a(T985I) conditional knock in mouse model for X-linked distal hereditary motor neuropathy (dHMNX)
Gonzalo Perez-Siles, Adrienne Grant, Melina Ellis, et al.
American Journal of Human Genetics
|
February 23, 2010
Missense mutations in the copper transporter gene ATP7A cause X-linked distal hereditary motor neuropathy
Marina L Kennerson, Garth A Nicholson, Stephen G Kaler, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
June 6, 2014
Oral treatment with Cu(II)(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis
Blaine R Roberts, Nastasia K H Lim, Erin J McAllum, et al.
Page
of 5