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Current Rheumatology Reports|December 6, 2014
Rotator cuff biology and biomechanics: a review of normal and pathological conditionsJulianne Huegel, Alexis A Williams, Louis J Soslowsky
Annals of Biomedical Engineering|January 7, 2021
Liquid Poly-N-acetyl Glucosamine (sNAG) Improves Achilles Tendon Healing in a Rat ModelCourtney A Nuss, Julianne Huegel, Sergio Finkielsztein, et al.
The American Journal of Pathology|April 12, 2015
Heparanase stimulates chondrogenesis and is up-regulated in human ectopic cartilage: a mechanism possibly involved in hereditary multiple exostosesJulianne Huegel, Motomi Enomoto-Iwamoto, Federica Sgariglia, et al.
Annals of Medicine and Surgery (2012)|September 4, 2023
Neptune versus canister-based surgical waste management in Europe: results of the GREEN studySarah Baker, Julie Clark, Angels B Pla, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|July 4, 2013
Heparan sulfate in skeletal development, growth, and pathology: the case of hereditary multiple exostosesJulianne Huegel, Federica Sgariglia, Motomi Enomoto-Iwamoto, et al.
Research Square|October 27, 2023
Simultaneous Inhibition of PI3K and PAK in Preclinical Models of Neurofibromatosis Type 2-related SchwannomatosisCristina Fernandez-Valle, Anna Nagel, Julianne Huegel, et al.
Oncogene|February 9, 2024
Simultaneous inhibition of PI3K and PAK in preclinical models of neurofibromatosis type 2-related schwannomatosisAnna Nagel, Julianne Huegel, Alejandra Petrilli, et al.
Developmental Biology|March 6, 2013
Perichondrium phenotype and border function are regulated by Ext1 and heparan sulfate in developing long bones: a mechanism likely deranged in Hereditary Multiple ExostosesJulianne Huegel, Christina Mundy, Federica Sgariglia, et al.
Annals of Biomedical Engineering|November 4, 2020
Chronic Nicotine Exposure Minimally Affects Rat Supraspinatus Tendon Properties and Bone MicrostructureJulianne Huegel, Courtney A Nuss, Peter Y W Chan, et al.
Bone|August 21, 2013
Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient miceFederica Sgariglia, Maria Elena Candela, Julianne Huegel, et al.
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