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Biochemical and Biophysical Research Communications|September 22, 2018
Truncated dystrophin ameliorates the dystrophic phenotype of mdx mice by reducing sarcolipin-mediated SERCA inhibitionJun Tanihata, Tetsuya Nagata, Naoki Ito, et al.Bioorganic & Medicinal Chemistry Letters|December 16, 2018
Synthesis of 2'-O-(N-methylcarbamoylethyl) 5-methyl-2-thiouridine and its application to splice-switching oligonucleotidesYoshiaki Masaki, Keishi Yamamoto, Takeshi Inde, et al.Journal of Nutritional Science and Vitaminology|July 16, 2009
Acute effects of dihydrocapsaicin and capsaicin on the distribution of white blood cells in ratsShunta Akimoto, Jun Tanihata, Fuuun Kawano, et al.Journal of Nutritional Science and Vitaminology|May 14, 2009
Zinc-deficiency induced changes in the distribution of rat white blood cellsYui Someya, Jun Tanihata, Shogo Sato, et al.Molecular Therapy. Nucleic Acids|February 4, 2015
Long-term efficacy of systemic multiexon skipping targeting dystrophin exons 45-55 with a cocktail of vivo-morpholinos in mdx52 miceYusuke Echigoya, Yoshitsugu Aoki, Bailey Miskew, et al.Plos One|July 30, 2013
Mutation types and aging differently affect revertant fiber expansion in dystrophic mdx and mdx52 miceYusuke Echigoya, Joshua Lee, Merryl Rodrigues, et al.International Journal of Molecular Sciences|October 19, 2016
Endogenous Multiple Exon Skipping and Back-Splicing at the DMD Mutation HotspotHitoshi Suzuki, Yoshitsugu Aoki, Toshiki Kameyama, et al.Journal of Applied Physiology (Bethesda, Md. : 1985)|October 29, 2020
Thiamine treatment preserves cardiac function against ischemia injury via maintaining mitochondrial size and ATP levelsYuki Yamada, Yoichiro Kusakari, Munetoshi Akaoka, et al.Proceedings of the National Academy of Sciences of the United States of America|August 8, 2012
Bodywide skipping of exons 45-55 in dystrophic mdx52 mice by systemic antisense deliveryYoshitsugu Aoki, Toshifumi Yokota, Tetsuya Nagata, et al.Proceedings of the National Academy of Sciences of the United States of America|August 21, 2025
RyR1-mediated Ca2+-induced Ca2+ release plays a negligible role in excitation-contraction coupling of normal skeletal muscleTakuya Kobayashi, Toshiko Yamazawa, Nagomi Kurebayashi, et al.Pageof 4