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Nature Neuroscience
|
April 2, 2013
FIP200 is required for maintenance and differentiation of postnatal neural stem cells
Chenran Wang, Chun-Chi Liang, Z Christine Bian, et al.
Molecular and Cellular Biology
|
February 21, 2008
Spatial interplay between PIASy and FIP200 in the regulation of signal transduction and transcriptional activity
Nadine Martin, Klaus Schwamborn, Henning Urlaub, et al.
British Journal of Cancer
|
April 28, 2020
Targeted therapy for mTORC1-driven tumours through HDAC inhibition by exploiting innate vulnerability of mTORC1 hyper-activation
Fuchun Yang, Shaogang Sun, Chenran Wang, et al.
The Journal of Cell Biology
|
October 4, 2006
Role of FIP200 in cardiac and liver development and its regulation of TNFalpha and TSC-mTOR signaling pathways
Boyi Gan, Xu Peng, Tamas Nagy, et al.
The Journal of Cell Biology
|
July 27, 2005
Identification of FIP200 interaction with the TSC1-TSC2 complex and its role in regulation of cell size control
Boyi Gan, Zara K Melkoumian, Xiaoyang Wu, et al.
The Journal of Cell Biology
|
June 10, 2010
Role of kinase-independent and -dependent functions of FAK in endothelial cell survival and barrier function during embryonic development
Xiaofeng Zhao, Xu Peng, Shaogang Sun, et al.
The Journal of Biological Chemistry
|
September 12, 2002
EphB1 associates with Grb7 and regulates cell migration
Dong Cho Han, Tang-Long Shen, Hui Miao, et al.
Breast Cancer Research : BCR
|
July 1, 2020
Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Ritama Paul, Ming Luo, Xueying Mo, et al.
American Journal of Physiology. Cell Physiology
|
June 30, 2021
Heterogeneity within molecular subtypes of breast cancer
Kevin M Turner, Syn Kok Yeo, Tammy M Holm, et al.
Breast Cancer Research : BCR
|
June 5, 2020
FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Ritama Paul, Ming Luo, Xueying Mo, et al.
Page
of 20
Search research articles
Search
Showing results (61-70 of 195) with videos related to
Sort By:
Page
of 20
Nature Neuroscience
|
April 2, 2013
FIP200 is required for maintenance and differentiation of postnatal neural stem cells
Chenran Wang, Chun-Chi Liang, Z Christine Bian, et al.
Molecular and Cellular Biology
|
February 21, 2008
Spatial interplay between PIASy and FIP200 in the regulation of signal transduction and transcriptional activity
Nadine Martin, Klaus Schwamborn, Henning Urlaub, et al.
British Journal of Cancer
|
April 28, 2020
Targeted therapy for mTORC1-driven tumours through HDAC inhibition by exploiting innate vulnerability of mTORC1 hyper-activation
Fuchun Yang, Shaogang Sun, Chenran Wang, et al.
The Journal of Cell Biology
|
October 4, 2006
Role of FIP200 in cardiac and liver development and its regulation of TNFalpha and TSC-mTOR signaling pathways
Boyi Gan, Xu Peng, Tamas Nagy, et al.
The Journal of Cell Biology
|
July 27, 2005
Identification of FIP200 interaction with the TSC1-TSC2 complex and its role in regulation of cell size control
Boyi Gan, Zara K Melkoumian, Xiaoyang Wu, et al.
The Journal of Cell Biology
|
June 10, 2010
Role of kinase-independent and -dependent functions of FAK in endothelial cell survival and barrier function during embryonic development
Xiaofeng Zhao, Xu Peng, Shaogang Sun, et al.
The Journal of Biological Chemistry
|
September 12, 2002
EphB1 associates with Grb7 and regulates cell migration
Dong Cho Han, Tang-Long Shen, Hui Miao, et al.
Breast Cancer Research : BCR
|
July 1, 2020
Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Ritama Paul, Ming Luo, Xueying Mo, et al.
American Journal of Physiology. Cell Physiology
|
June 30, 2021
Heterogeneity within molecular subtypes of breast cancer
Kevin M Turner, Syn Kok Yeo, Tammy M Holm, et al.
Breast Cancer Research : BCR
|
June 5, 2020
FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Ritama Paul, Ming Luo, Xueying Mo, et al.
Page
of 20