Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Jun-ichi Abe

Showing results (151-160 of 160) with videos related to

Pageof 16
Sort By:
You have reached the last page of results.This site can display upto 160 results.
Circulation|December 28, 2018
Senescent Phenotype Induced by p90RSK-NRF2 Signaling Sensitizes Monocytes and Macrophages to Oxidative Stress in HIV-Positive IndividualsMeera V Singh, Sivareddy Kotla, Nhat-Tu Le, et al.
JCI Insight|April 5, 2019
MAGI1 as a link between endothelial activation and ER stress drives atherosclerosisJun-Ichi Abe, Kyung Ae Ko, Sivareddy Kotla, et al.
Frontiers in Cardiovascular Medicine|September 15, 2023
Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transitionMinh T H Nguyen, Masaki Imanishi, Shengyu Li, et al.
Antioxidants (Basel, Switzerland)|December 30, 2025
SHP2: A Redox-Sensitive Regulator Linking Immune Checkpoint Inhibitor Therapy to Cancer Treatment and Vascular RiskSilvia Fernanda López Moreno, Stefania Assunto Lenz, Bernardo Casso-Chapa, et al.
Circulation Research|June 2, 2023
An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and AtherosclerosisJun-Ichi Abe, Masaki Imanishi, Shengyu Li, et al.
Frontiers in Cardiovascular Medicine|October 2, 2023
Premature senescence and cardiovascular disease following cancer treatments: mechanistic insightsAshita Jain, Diego Casanova, Alejandra Valdivia Padilla, et al.
Redox Biology|October 7, 2021
Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiationSivareddy Kotla, Aijun Zhang, Masaki Imanishi, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 16, 2025
Reevaluating Anti-Inflammatory Therapy: Targeting Senescence to Balance Anti-Cancer Efficacy and Vascular DiseaseBernardo Casso-Chapa, Norma Alicia Vazquez González, Nhat-Tu Le, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
Downregulation of LATS1/2 Drives Endothelial Senescence-Associated Stemness (SAS) and Atherothrombotic Lesion FormationSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
Circulation Research|May 27, 2026
LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque ThrombosisSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
Pageof 16

Showing results (151-160 of 160) with videos related to

Sort By:
Pageof 16
You have reached the last page of results.This site can display upto 160 results.
Circulation|December 28, 2018
Senescent Phenotype Induced by p90RSK-NRF2 Signaling Sensitizes Monocytes and Macrophages to Oxidative Stress in HIV-Positive IndividualsMeera V Singh, Sivareddy Kotla, Nhat-Tu Le, et al.
JCI Insight|April 5, 2019
MAGI1 as a link between endothelial activation and ER stress drives atherosclerosisJun-Ichi Abe, Kyung Ae Ko, Sivareddy Kotla, et al.
Frontiers in Cardiovascular Medicine|September 15, 2023
Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transitionMinh T H Nguyen, Masaki Imanishi, Shengyu Li, et al.
Antioxidants (Basel, Switzerland)|December 30, 2025
SHP2: A Redox-Sensitive Regulator Linking Immune Checkpoint Inhibitor Therapy to Cancer Treatment and Vascular RiskSilvia Fernanda López Moreno, Stefania Assunto Lenz, Bernardo Casso-Chapa, et al.
Circulation Research|June 2, 2023
An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and AtherosclerosisJun-Ichi Abe, Masaki Imanishi, Shengyu Li, et al.
Frontiers in Cardiovascular Medicine|October 2, 2023
Premature senescence and cardiovascular disease following cancer treatments: mechanistic insightsAshita Jain, Diego Casanova, Alejandra Valdivia Padilla, et al.
Redox Biology|October 7, 2021
Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiationSivareddy Kotla, Aijun Zhang, Masaki Imanishi, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 16, 2025
Reevaluating Anti-Inflammatory Therapy: Targeting Senescence to Balance Anti-Cancer Efficacy and Vascular DiseaseBernardo Casso-Chapa, Norma Alicia Vazquez González, Nhat-Tu Le, et al.
Biorxiv : the Preprint Server for Biology|July 16, 2025
Downregulation of LATS1/2 Drives Endothelial Senescence-Associated Stemness (SAS) and Atherothrombotic Lesion FormationSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
Circulation Research|May 27, 2026
LATS1/2-CD38 Metabolic Rewiring Links Senescence to Intraplaque ThrombosisSivareddy Kotla, Jonghae Lee, Kyung Ae Ko, et al.
Pageof 16