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Plos One|July 1, 2016
DNA Hypermethylation of CREB3L1 and Bcl-2 Associated with the Mitochondrial-Mediated Apoptosis via PI3K/Akt Pathway in Human BEAS-2B Cells Exposure to Silica NanoparticlesYang Zou, Qiuling Li, Lizhen Jiang, et al.Plos One|April 18, 2013
Acute toxicity of amorphous silica nanoparticles in intravenously exposed ICR miceYang Yu, Yang Li, Wen Wang, et al.Environment & Health (Washington, D.C.)|February 27, 2025
Subnational Evidence for the Attributable Burden of Respiratory Infections in China's Population under 20: Challenges from Particulate Matter PollutionJunchao Duan, Ruiyang Ding, Menglong Li, et al.Toxicology Research|August 10, 2018
Autophagy and autophagy dysfunction contribute to apoptosis in HepG2 cells exposed to nanosilicaYongbo Yu, Junchao Duan, Yang Yu, et al.The Science of the Total Environment|June 5, 2021
The relationship between exposure to PM2.5 and atrial fibrillation in older adults: A systematic review and meta-analysisFenghong Wang, Xapkat Ahat, Qingqing Liang, et al.The Science of the Total Environment|May 16, 2021
PM2.5 exposure exaggerates the risk of adverse birth outcomes in pregnant women with pre-existing hyperlipidemia: Modulation role of adipokines and lipidomeJingyi Zhang, Gongbo Chen, Shuang Liang, et al.Redox Biology|August 23, 2021
The mitochondria-targeted antioxidant MitoQ attenuated PM2.5-induced vascular fibrosis via regulating mitophagyRuihong Ning, Yang Li, Zhou Du, et al.Journal of Hazardous Materials|March 4, 2014
Silica nanoparticles induce autophagy and autophagic cell death in HepG2 cells triggered by reactive oxygen speciesYongbo Yu, Junchao Duan, Yang Yu, et al.Environmental Science and Pollution Research International|March 23, 2017
Genome-wide transcriptional analysis of cardiovascular-related genes and pathways induced by PM2.5 in human myocardial cellsLin Feng, Xiaozhe Yang, Collins Otieno Asweto, et al.Environmental Science and Pollution Research International|September 5, 2016
Co-exposure to amorphous silica nanoparticles and benzo[a]pyrene at low level in human bronchial epithelial BEAS-2B cellsJing Wu, Yanfeng Shi, Collins Otieno Asweto, et al.Pageof 16