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Bone|November 21, 2024
A NOTCH2 pathogenic variant and HES1 regulate osteoclastogenesis in induced pluripotent stem cellsErnesto Canalis, Lauren Schilling, Emily Denker, et al.The Journal of Biological Chemistry|October 21, 2023
NOTCH2 sensitizes the chondrocyte to the inflammatory response of tumor necrosis factor αErnesto Canalis, Jungeun Yu, Vijender Singh, et al.The Journal of Biological Chemistry|March 28, 2021
Activation of Notch3 in osteoblasts/osteocytes causes compartment-specific changes in bone remodelingErnesto Canalis, Stefano Zanotti, Lauren Schilling, et al.Plos One|May 10, 2022
Use of antisense oligonucleotides to target Notch3 in skeletal cellsErnesto Canalis, Michele Carrer, Tabitha Eller, et al.Plos One|January 3, 2025
A NOTCH3 pathogenic variant influences osteogenesis and can be targeted by antisense oligonucleotides in induced pluripotent stem cellsErnesto Canalis, Jungeun Yu, Lauren Schilling, et al.The Journal of Biological Chemistry|July 26, 2018
The lateral meningocele syndrome mutation causes marked osteopenia in miceErnesto Canalis, Jungeun Yu, Lauren Schilling, et al.The Journal of Biological Chemistry|January 30, 2020
Antisense oligonucleotides targeting Notch2 ameliorate the osteopenic phenotype in a mouse model of Hajdu-Cheney syndromeErnesto Canalis, Tamar R Grossman, Michele Carrer, et al.The American Journal of Pathology|October 18, 2017
The Hajdu Cheney Mutation Is a Determinant of B-Cell Allocation of the Splenic Marginal ZoneJungeun Yu, Stefano Zanotti, Bhavita Walia, et al.Plos One|October 31, 2019
A PDGFRβ-PI3K signaling axis mediates periosteal cell activation during fracture healingLaura Doherty, Jungeun Yu, Xi Wang, et al.Bone|November 4, 2025
A novel variant of NOTCH2 causes skeletal fragilityErnesto Canalis, Jungeun Yu, Emily Denker, et al.Pageof 4