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The EMBO Journal|November 18, 2022
LUBAC and NF-κB trigger a nuclear response from mitochondriaJunsheng Chen, Thomas SimmenContact (Thousand Oaks (Ventura County, Calif.))|June 27, 2023
Post-Translational Modification of Cysteines: A Key Determinant of Endoplasmic Reticulum-Mitochondria Contacts (MERCs)Arthur Bassot, Junsheng Chen, Thomas SimmenCell Calcium|October 29, 2024
ER stress as a sentinel mechanism for ER Ca<sup>2+</sup> homeostasisTadashi Makio, Junsheng Chen, Thomas SimmenCells|August 7, 2021
Amyotrophic Lateral Sclerosis (ALS): Stressed by Dysfunctional Mitochondria-Endoplasmic Reticulum Contacts (MERCs)Junsheng Chen, Arthur Bassot, Fabrizio Giuliani, et al.Expert Opinion on Therapeutic Targets|March 12, 2011
Hax-1: a regulator of calcium signaling and apoptosis progression with multiple roles in human diseaseThomas SimmenCell Calcium|June 17, 2017
Endoplasmic reticulum chaperones tweak the mitochondrial calcium rheostat to control metabolism and cell deathTomas Gutiérrez, Thomas SimmenFrontiers in Oncology|November 12, 2014
Endoplasmic reticulum chaperones and oxidoreductases: critical regulators of tumor cell survival and immunorecognitionTomás Gutiérrez, Thomas SimmenContact (Thousand Oaks (Ventura County, Calif.))|July 29, 2024
Not So Rare: Diseases Based on Mutant Proteins Controlling Endoplasmic Reticulum-Mitochondria Contact (MERC) TetheringTadashi Makio, Thomas SimmenAdvances in Experimental Medicine and Biology|August 18, 2017
Organelle Communication at Membrane Contact Sites (MCS): From Curiosity to Center Stage in Cell Biology and Biomedical ResearchThomas Simmen, Mitsuo TagayaJournal of Cell Science|May 8, 2025
A lipid in transit - the journey of cholesterol into the heart of mitochondrial researchThomas Simmen, Luca PellegriniPageof 24