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Juozas A Zavadzkas

Showing results (1-10 of 13) with videos related to

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The Journal of Surgical Research|September 25, 2010
Discordant activation of gene promoters for matrix metalloproteinases and tissue inhibitors of the metalloproteinases following myocardial infarctionRupak Mukherjee, Jonathan M Snipes, Stuart M Saunders, et al.
The Journal of Biological Chemistry|July 21, 2010
Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarctionFrancis G Spinale, Rupak Mukherjee, Juozas A Zavadzkas, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 8, 2009
Calpain inhibition preserves myocardial structure and function following myocardial infarctionSanthosh K Mani, Sundaravadivel Balasubramanian, Juozas A Zavadzkas, et al.
The Journal of Thoracic and Cardiovascular Surgery|March 12, 2010
Cellular phenotype transformation occurs during thoracic aortic aneurysm developmentJeffrey A Jones, Juozas A Zavadzkas, Eileen I Chang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 27, 2010
Alterations in membrane type-1 matrix metalloproteinase abundance after the induction of thoracic aortic aneurysm in a murine modelJeffrey A Jones, Jean Marie Ruddy, Shenikqua Bouges, et al.
Circulation. Heart Failure|October 8, 2009
Cardiac-restricted overexpression of membrane type-1 matrix metalloproteinase in mice: effects on myocardial remodeling with agingFrancis G Spinale, G Patricia Escobar, Rupak Mukherjee, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 18, 2010
Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activityRupak Mukherjee, Juozas A Zavadzkas, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 12, 2008
Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging miceJuozas A Zavadzkas, Rebecca A Plyler, Shenikqua Bouges, et al.
American Journal of Physiology. Heart and Circulatory Physiology|June 14, 2011
Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodelingJuozas A Zavadzkas, Rupak Mukherjee, William T Rivers, et al.
Journal of Cardiovascular Pharmacology|October 9, 2008
Aprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor IMichel J Sabbagh, J Michael Looper, Juozas A Zavadzkas, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
The Journal of Surgical Research|September 25, 2010
Discordant activation of gene promoters for matrix metalloproteinases and tissue inhibitors of the metalloproteinases following myocardial infarctionRupak Mukherjee, Jonathan M Snipes, Stuart M Saunders, et al.
The Journal of Biological Chemistry|July 21, 2010
Cardiac restricted overexpression of membrane type-1 matrix metalloproteinase causes adverse myocardial remodeling following myocardial infarctionFrancis G Spinale, Rupak Mukherjee, Juozas A Zavadzkas, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 8, 2009
Calpain inhibition preserves myocardial structure and function following myocardial infarctionSanthosh K Mani, Sundaravadivel Balasubramanian, Juozas A Zavadzkas, et al.
The Journal of Thoracic and Cardiovascular Surgery|March 12, 2010
Cellular phenotype transformation occurs during thoracic aortic aneurysm developmentJeffrey A Jones, Juozas A Zavadzkas, Eileen I Chang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 27, 2010
Alterations in membrane type-1 matrix metalloproteinase abundance after the induction of thoracic aortic aneurysm in a murine modelJeffrey A Jones, Jean Marie Ruddy, Shenikqua Bouges, et al.
Circulation. Heart Failure|October 8, 2009
Cardiac-restricted overexpression of membrane type-1 matrix metalloproteinase in mice: effects on myocardial remodeling with agingFrancis G Spinale, G Patricia Escobar, Rupak Mukherjee, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 18, 2010
Short-term disruption in regional left ventricular electrical conduction patterns increases interstitial matrix metalloproteinase activityRupak Mukherjee, Juozas A Zavadzkas, William T Rivers, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 12, 2008
Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging miceJuozas A Zavadzkas, Rebecca A Plyler, Shenikqua Bouges, et al.
American Journal of Physiology. Heart and Circulatory Physiology|June 14, 2011
Direct regulation of membrane type 1 matrix metalloproteinase following myocardial infarction causes changes in survival, cardiac function, and remodelingJuozas A Zavadzkas, Rupak Mukherjee, William T Rivers, et al.
Journal of Cardiovascular Pharmacology|October 9, 2008
Aprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor IMichel J Sabbagh, J Michael Looper, Juozas A Zavadzkas, et al.
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