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Research in Microbiology|May 15, 2004
When replication travels on damaged templates: bumps and blocks in the roadJustin Courcelle, Jerilyn J Belle, Charmain T CourcelleJournal of Molecular Biology|May 24, 2011
Escherichia coli Fpg glycosylase is nonrendundant and required for the rapid global repair of oxidized purine and pyrimidine damage in vivoBrandy J Schalow, Charmain T Courcelle, Justin CourcelleGenes|March 25, 2022
Recombination Mediator Proteins: Misnomers That Are Key to Understanding the Genomic Instabilities in CancerJustin Courcelle, Travis K Worley, Charmain T CourcelleJournal of Bacteriology|May 26, 2009
Nucleotide excision repair is a predominant mechanism for processing nitrofurazone-induced DNA damage in Escherichia coliKatherine R Ona, Charmain T Courcelle, Justin CourcelleJournal of Nucleic Acids|October 12, 2012
UvrD Participation in Nucleotide Excision Repair Is Required for the Recovery of DNA Synthesis following UV-Induced Damage in Escherichia coliKelley N Newton, Charmain T Courcelle, Justin CourcelleJournal of Molecular Biology|June 19, 2010
ATP binding, ATP hydrolysis, and protein dimerization are required for RecF to catalyze an early step in the processing and recovery of replication forks disrupted by DNA damageEmilie Michel-Marks, Charmain T Courcelle, Sergey Korolev, et al.Journal of Bacteriology|May 29, 2012
Cellular characterization of the primosome and rep helicase in processing and restoration of replication following arrest by UV-induced DNA damage in Escherichia coliCharmain T Courcelle, Allison J Landstrom, Brittany Anderson, et al.Genes to Cells : Devoted to Molecular & Cellular Mechanisms|December 31, 2020
UV-induced DNA damage disrupts the coordination between replication initiation, elongation and completionBrian M Wendel, Suzanne Hollingsworth, Charmain T Courcelle, et al.Journal of Bacteriology|May 29, 2007
Inactivation of the DnaB helicase leads to the collapse and degradation of the replication fork: a comparison to UV-induced arrestJerilyn J Belle, Andrew Casey, Charmain T Courcelle, et al.Mutation Research|September 11, 2008
RecA433 cells are defective in recF-mediated processing of disrupted replication forks but retain recBCD-mediated functionsQais Al-Hadid, Katherine Ona, Charmain T Courcelle, et al.Pageof 10