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Molecular Cell|February 1, 2020
Allosteric Regulation of BH3 Proteins in Bcl-xL Complexes Enables Switch-like Activation of BaxChristian Bogner, Justin Kale, Justin Pogmore, et al.Biochimica Et Biophysica Acta. Molecular Cell Research|October 6, 2024
CISD2 counteracts the inhibition of ER-mitochondrial calcium transfer by anti-apoptotic BCL-2Jens Loncke, Ian de Ridder, Justin Kale, et al.The Journal of Biological Chemistry|March 12, 2014
After embedding in membranes antiapoptotic Bcl-XL protein binds both Bcl-2 homology region 3 and helix 1 of proapoptotic Bax protein to inhibit apoptotic mitochondrial permeabilizationJingzhen Ding, Blaine H M Mooers, Zhi Zhang, et al.Frontiers in Pharmacology|April 9, 2026
VB-84922 is a small molecule that inhibits ER-to-golgi transport of SREBPs-SCAP complexesJ Jose Corbalan, Wiebke Schormann, Pranavi Jagadeesan, et al.Cell Death & Disease|September 18, 2020
BDA-366, a putative Bcl-2 BH4 domain antagonist, induces apoptosis independently of Bcl-2 in a variety of cancer cell modelsTamara Vervloessem, Binu K Sasi, Elena Xerxa, et al.The EMBO Journal|December 25, 2015
BH3-in-groove dimerization initiates and helix 9 dimerization expands Bax pore assembly in membranesZhi Zhang, Sabareesh Subramaniam, Justin Kale, et al.The EMBO Journal|June 8, 2021
An amphipathic Bax core dimer forms part of the apoptotic pore wall in the mitochondrial␣membraneFujiao Lv, Fei Qi, Zhi Zhang, et al.Cell Death and Differentiation|November 9, 2021
Bcl-xL acts as an inhibitor of IP3R channels, thereby antagonizing Ca2+-driven apoptosisNicolas Rosa, Hristina Ivanova, Larry E Wagner, et al.Pageof 2